<rdf:RDF xmlns:rdf="http://www.w3.org/1999/02/22-rdf-syntax-ns#" xmlns:dcterms="http://purl.org/dc/terms/">
<rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/49">
    <dcterms:title><![CDATA[Síndrome agudo de cauda equina por extrusión discal: presentación de 5 casos]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Serie de Casos]]></dcterms:description>
    <dcterms:abstract><![CDATA[El síndrome agudo de cauda equina (SACE) es una patología infrecuente, cuya principal causa es la hernia discal lumbar (HDL) extruída. Sin embargo, solo en un 2% de las extrusiones discales se genera un SACE. Otras etiologías menos frecuentes responden a tumores, traumas y complicaciones postquirúrgicas. <br />
Como resultado de la compresión de las raíces nerviosas distales al cono medular (cola de caballo o cauda equina), aparece un síndrome que consiste en dolor lumbar, radiculalgias uni o bilaterales, debilidad motora en los miembros inferiores, déficits sensitivos, y trastornos en la esfera sexual y esfinteriana. En raras ocasiones se encuentran todas estas manifestaciones, con distintas combinaciones de síntomas y signos. <br />
Algunos autores dividen al SACE en: incompleto (SACEI), cuando no hay retención urinaria ni fecal; y completo (SACEC), cuando aparece la retención. <br />
El diagnóstico se realiza con la clínica compatible y se confirma mediante estudios de imágenes, siendo la Resonancia Magnética el método más sensible y específico.<br />
En la bibliografía no existe consenso en cuanto al mejor momento para realizar la cirugía, ni cuál es el procedimiento quirúrgico de elección.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Alfredo Guiroy]]></dcterms:creator>
    <dcterms:creator><![CDATA[Martín Gagliardi]]></dcterms:creator>
    <dcterms:creator><![CDATA[Federico Fernández Molina]]></dcterms:creator>
    <dcterms:creator><![CDATA[Pablo Jalón]]></dcterms:creator>
    <dcterms:creator><![CDATA[Juan José Mezzadri]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Marcelo Platas]]></dcterms:publisher>
    <dcterms:date><![CDATA[Junio 2014]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[<p>&nbsp;</p>
<ol>
<li>Bartels RHMA, deVries J. Hemi-cauda equina syndrome from herniated lumbar disc: a neurosurgical emergency? Can J NeurolSci 1996;23:296&ndash;9.</li>
<li>Buchner M, Schiltenwolf M. Cauda equina syndrome caused by intervertebral lumbar disk prolapse: mid-term results of 22 patients and literature review. Orthopedics 2002;25:727&ndash;31.</li>
<li>Dinning T and Schaeffer H. Discogenic compression of the cauda equina: A surgical emergency. Aust N Z J Surg 63:927&ndash;934. 1993.</li>
<li>Dinning TAR, Schaeffer HR. Discogenic compression of cauda equina: a surgical emergency. Aust NZ J Surg1993;63:927&ndash;34.</li>
<li>Gleave JRW, Macfarlane R. Prognosis of recovery of bladder function following lumbar central disc prolapse. Br J Neurosurg1990; 4:205&ndash;10.</li>
<li>Henriques T, Olerud C, Petre&acute; n-Mallmin M, et al. Cauda equina syndrome as a postoperative complication infive patients operated for lumbar disc herniation. Spine 2001;26:293&ndash;7.</li>
<li>Kostuik JP. Controversies in cauda equina syndrome and lumbar disk herniations. CurrOpinOrthop. 1993;4:125&ndash;8.</li>
<li>O&rsquo;Laoire SA, Crockard HA, Thomas DG. Prognosis for sphincter recovery after operation for operation for cauda equina compression owing to lumbar disc prolapse. Br MedJ 1981;282:1852&ndash; 4.</li>
<li>Shapiro S. Cauda equina syndrome secondary to lumbardisc herniation. Neurosurgery 1993;32:743&ndash;7.</li>
<li>Shapiro S. Medical realities of cauda equina syndrome secondary to lumbar disc herniation. Spine 2000;25:348&ndash;52.</li>
</ol>]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/1210">
    <dcterms:title><![CDATA[Síndrome de Cola de Caballo asociado a tratamiento quiropráctico: Reporte de 3 casos y revisión de la literatura]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[RESUMEN<br />
El síndrome de cola de caballo (SCC) es una urgencia quirúrgica poco frecuente con una incidencia estimada de hasta 1,8 casos por millón de habitantes, producida por la compresión de las raíces nerviosas en el extremo inferior del canal espinal. La manipulación espinal puede desempeñar un papel etiogénico, provocando la movilización y extrusión del disco. El diagnóstico temprano y el tratamiento oportuno son cruciales, ya que el pronóstico suele ser desfavorable si el tratamiento quirúrgico se retrasa produciendo un daño neurológico permanente.<br />
El objetivo de este trabajo es identificar los potenciales factores de riesgo para la manipulación espinal y optimizar esta práctica, evitando así posibles complicaciones derivadas del tratamiento quiropráctico. Presentamos 3 casos de SCC, observados y tratados en nuestro centro, en los que se sugiere una estrecha relación entre la manipulación espinal quiropráctica y la aparición de dicho síndrome. Tras realizarles una RM en la que se observó una hernia discal L5-S1 causante del SCC, los 3 pacientes fueron tratados quirúrgicamente de forma urgente.<br />
Los casos presentados demostraron la existencia de una asociación patogénica entre la manipulación espinal y el desarrollo del SCC, al producirse dicho síndrome en las horas siguientes a la manipulación debida a la protusión abrupta de un disco demostrado por RM.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Joaquin Andrés Andermatten]]></dcterms:creator>
    <dcterms:creator><![CDATA[Nicolás Sampron Lebed]]></dcterms:creator>
    <dcterms:creator><![CDATA[ Jose Undabeitia Huertas<br />
]]></dcterms:creator>
    <dcterms:creator><![CDATA[Sergio Torres Bayona]]></dcterms:creator>
    <dcterms:creator><![CDATA[Patricia Torres Perez]]></dcterms:creator>
    <dcterms:creator><![CDATA[Mariano Arrazola Schlamilch]]></dcterms:creator>
    <dcterms:creator><![CDATA[Enrique Urculo Bareño]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Mariano Socolovsky]]></dcterms:publisher>
    <dcterms:date><![CDATA[Agosto 2018]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[BIBLIOGRAFÍA<br />
<br />
Ahn UM, Ahn Nu, Buchowski MS, Garrett ES, Sieber AN, KostuikJP. Cauda equina syndrome secondary to lumbar disc herniation: a meta-analysis of surgical outcomes. Spine. 2000. 25:1515-22.<br />
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<br />
Boucher P, Robidoux S. Lumbar disc herniation and cauda equina syndrome following spinal manipulative therapy: a review of six court decisions in Canada. J Forensic Legal Med. 2014. 22:159–169. <br />
<br />
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Bydon M, Lin JA, de la Garza-Ramos R, Macki M, Kosztowski T, Sciubba D, et al. Time to surgery and outcomes in cauda equina syndrome: An analysis of 45 cases. World Neurosurg. 2015, pi: S1878-8750(15)01758-1.<br />
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Celik EC, Kabatas S, Karatas M. Atypical presentation of cauda equina syndrome secondary to lumbar disc herniation. J Back Musculoskelet Rehabil 2012. 25:1-3.<br />
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Coulter ID. Efficacy and risks of chiropractic manipulation: what does the evidence suggest? Integ Med 1998; 1:61–6.<br />
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Dupeyron A, Vautravers P, Lecocq J, Isner-Horobeti ME. Complications following vertebral manipulation—a survey of aFrench region physicians. Ann Readapt Med Phys 2002; 46:33–40. <br />
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Ernst E. Prospective investigations into the safety of spinal manipulation. J Pain Symptom Manag. 2001. 21:238–242.<br />
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Gouveia LO, Castanho P, Ferreira JJ. Safety of chiropractic interventions: a systematic review. Spine (Phila Pa 1976) 2009; 34 (11):E405-13.<br />
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Haldeman S, Rubinstein SM. Cauda equina syndrome in patients undergoing manipulation of the lumbar spine. Spine. 1992. 17:1469–1473.<br />
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Hebert J.J., Stomski N.J., French S.D., Rubinstein S.M. Serious Adverse Events and Spinal Manipulative Therapy of the Low Back Region: A Systematic Review of Cases. Journal of Manipulative and Physiological Therapeutics, 2015. 38 (9), pp. 677-691.<br />
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Kholes SS, Kholes DA, Karp AP, Erlich VM. Time dependent surgical outcomes following cauda equina syndrome diagnosis: coments on a meta-analisis. Spine. 2004. 29:1281-7.<br />
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Lisi AJ, Holmes EJ, Ammendolia C. High-velocity low amplitude spinal manipulation for symptomatic lumbar disk disease: a systematic review of the literature. J ManipPhysiolTher. 2005. 28:429–442.<br />
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Malmivaara A, Pohjola R. Cauda equina syndrome caused by chiropraxis on a patient previously free of lumbar spine symptoms. Lancet 1982; 2:986–7.<br />
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Oliphant D. Safety of spinal manipulation in the treatment of lumbar disk herniations: a systematic review and risk assessment. J ManipPhysiolTher. 2004. 27:197–210.<br />
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Olivero WC, Wang H, Hanigan WC, Henderson JP, Tracy. PT, Elwood PW, et al. Cauda Equina Syndrome from lumbar disc herniations. J Spinal Disord Tech 2009; 22:202-6.<br />
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Oppenheim JS, Spitzer DE, Segal DH. Nonvascular complications following spinal manipulation. Spine J. 2005; 5(6):660–6.<br />
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Schmidley JW, Koch T. The noncerebrovascular complications of chiropractic manipulation. Neurology 1984. 34:684–5.<br />
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Silver JR. The earliest case of cauda equina syndrome caused by manipulation of the lumbar spine under a general anaesthetic. Spinal Cord. 2001. 39:51–53.<br />
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Swait G, Finch R. What are the risks of manual treatment of the spine? A scoping review for clinicians. Chiropractic &amp; Manual Therapies. 2017; 25:37.<br />
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Tamburrelli FC, Genitiempo M, Logroscino CA. Cauda equina syndrome and spine manipulation: case report and review of the literature. Eur Spine J 2011; 20 Suppl 1:S128–S131.<br />
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Undabeitia J, et al. Síndrome de cola de caballo tras tratamiento quiropráctico. Neurocirugia. 2016.Volume 27, Issue 3, May–June 2016, Pages 151–153. ]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/271">
    <dcterms:title><![CDATA[Síndrome de Grisel en el adulto e inestabilidad cráneo cervical. Odontoidectomía endoscópica transnasal y fijación occípito-cervical. Reporte de caso y revisión de literatura]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Reporte de Caso]]></dcterms:description>
    <dcterms:abstract><![CDATA[Introducción: La patología de la unión cráneo-cervical es poco frecuente en la práctica neuroquirúrgica diaria. En general la mayor parte de estas lesiones son de origen traumático o reumatológico. La inestabilidad atlanto-axial de origen inflamatorio (Síndrome de Grisel) es una rara entidad de la que solo se han reportado 16 casos adultos en la literatura. Esta patología se caracteriza por el desarrollo de una lesión osteolítica a nivel de la articulación atlanto-axoidea posterior a un evento infeccioso, generalmente de las vías aéreas superiores. <br />
Caso clínico: Presentamos el caso de una paciente de 76 años quien concurre en consulta por un cuadro clínico de inestabilidad cráneo espinal secundaria a una lesión osteolítica, con compromiso de C1 y C2. La sintomatología comenzó posteriormente a un proceso infeccioso respiratorio. Se realizó una fijación occípito-cervical posterior y una Odontoidectomía con descompresión anterior por vía endoscópica transnasal. La paciente evolucionó con resolución completa de los síntomas. Los cultivos fueron negativos y el estudio de anatomía patológica concluyó cambios inflamatorios inespecíficos.<br />
Conclusión: Hasta hace algunos pocos años la única opción para abordar esta patología era la vía transoral con técnica microquirúrgica. Hoy en día la endoscopía nos brinda muchas ventajas técnicas por lo que constituye una opción a tener en cuenta en la planificación de los abordajes a la unión cráneo-cervical.<br />
]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Ariel Estramiana]]></dcterms:creator>
    <dcterms:creator><![CDATA[Julieta Gómez]]></dcterms:creator>
    <dcterms:creator><![CDATA[Roberto Herrera]]></dcterms:creator>
    <dcterms:creator><![CDATA[José Luis Ledesma]]></dcterms:creator>
    <dcterms:creator><![CDATA[José Pablo]]></dcterms:creator>
    <dcterms:creator><![CDATA[Julián Pastore]]></dcterms:creator>
    <dcterms:creator><![CDATA[Héctor Rojas]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Mariano Socolovsky]]></dcterms:publisher>
    <dcterms:date><![CDATA[Julio 2017]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/101">
    <dcterms:title><![CDATA[Síndrome de Hakim Adams: evaluación de la calidad de vida luego de la cirugía]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[<p><strong>RESUMEN</strong><br /> <strong>Introducci&oacute;n:</strong> El s&iacute;ndrome de Hakim Adams, o hidrocefalia normotensiva, es un cuadro progresivo que se caracteriza por presentar alteraci&oacute;n de la marcha, trastornos esfinterianos y demencia, y que afecta principalmente a pacientes a&ntilde;osos. Si bien existen publicaciones sobre los resultados del tratamiento quir&uacute;rgico, estos trabajos suelen evaluar la mejor&iacute;a sintom&aacute;tica pero no la calidad de vida de los pacientes. <br /> <strong>Objetivo: </strong>Conocer el impacto del tratamiento en pacientes con s&iacute;ndrome de Hakim Adams tratados con derivaci&oacute;n de LCR. <br /> <strong>Material y M&eacute;todos:</strong> Realizamos un estudio retrospectivo de una serie de 8 pacientes. Se realiz&oacute; una encuesta para la evaluaci&oacute;n preoperatoria y postoperatoria; con un tiempo transcurrido no menor a 6 meses luego del procedimiento. Se utiliz&oacute; la escala de Barthel para la funcionalidad, y una escala num&eacute;rica simple para la calidad de vida. <br /> Resultados: Previo a la cirug&iacute;a, 7 casos presentaban alteraciones de la marcha y/o problemas para deambular; 6 casos alteraciones esfinterianas; y 5 casos alg&uacute;n tipo de alteraci&oacute;n neurocognitiva. Luego de la cirug&iacute;a, todos los pacientes refirieron un cambio significativo en la calidad de vida; y tambi&eacute;n en la funcionalidad, logrando 6 pacientes una independencia absoluta. <br /> <strong>Conclusi&oacute;n: </strong>Aunque nuestra comunicaci&oacute;n tiene las debilidades de una serie peque&ntilde;a de casos analizados en forma retrospectiva, los resultados son concluyentes en cuanto a mejora en la calidad de vida y funcionalidad en el postoperatorio. Creemos que esta modalidad de evaluaci&oacute;n puede ayudar a los pacientes y/o familiares de pacientes en el proceso de toma decisiones del tratamiento quir&uacute;rgico.</p>
<p><strong>Palabras Claves:</strong> S&iacute;ndrome de Hakim Adams; Hidrocefalia Normotensiva; Demencia; Trastorno de la Marcha; Derivaci&oacute;n de L&iacute;quido Cefalorraqu&iacute;deo; Calidad de Vida</p>]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Alejandra T. Rabadan]]></dcterms:creator>
    <dcterms:creator><![CDATA[Román Martín González]]></dcterms:creator>
    <dcterms:creator><![CDATA[María Victoria García Fernández]]></dcterms:creator>
    <dcterms:creator><![CDATA[Roberto Freue]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Jaime Rimoldi]]></dcterms:publisher>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[<ol>
<li>Adams RD, Fisher CM, Hakim S, et al. Symptomatic occult hydrocepahalus with &ldquo;normal&rdquo; cerebrospinal &ndash;fluid pressure: A treatable syndrome. N Eng J Med 1965; 273: 117-126.</li>
<li>Bergsneider M, Black P, Klinge P, et al. Surgical management of idiopathic normal-pressure hydrocephalus. Neurosurgery (Suppl) 2005; 57 (3): S2-29-S2-39.</li>
<li>Duikerke A, Williams MA, Rigamonti D, et al. Cognitive recovery in idiopathic normal pressure hydrocephalus after shunt. Cogn Behav Neurol 2004; 17 (3): 179-84.</li>
<li>Ferri, C.P., Prince, M., Brayne, C., et al. Global prevalence of dementia: a Delphi consensus study. Lancet 2005; 366 (9503): 2112-2117.</li>
<li>Freter S, Bergman H, Gold S, Chertkow H, Clarfield M. Prevalence of potencially reversible dementias and actual reversibility in a memory clinic cohort. CMAJ 1998; 159: 657-662.</li>
<li>Kahlon B, Sjunnesson J, Rehncrona S. Long-term outcome in patients with suspected normal pressure hydrocepahalus. Neurosurgery 2007; 60 (2): 327-32.</li>
<li>Kilic K, Czorny A, Auque J, et al. Predicting the outcome of shunt surgery in normal pressure hydrocephalus. J Clin Neurosci 2007; 14 (8): 729-36.</li>
<li>Klinge P, Marmarou A, Bergsneider M, et al. Outcome of shunting in idiopathic normal-pressure hydrocephalus and the value of outcome assesment in shunted patients. Neurosurgery (Suppl) 2005; 57 (3): S2-40-S2-52.</li>
<li>Marmarou A, Young HF, Aygok GA. Estimated incidence of normal pressure hydrocephalus and shunt outcome in patients residing in assisted-living and extended-care facilities. Neurosurg Focus 2007; 22(4):E1.</li>
<li>Pedro-Cuesta, Virues-Ortega, J., Vega, S., et al. Prevalence of dementia and major dementia subtypes in Spanish populations: a reanalysis of dementia prevalence surveys, 1990-2008. BMC Neurology 2009; 9 (55): 1-9.</li>
<li>Rabad&aacute;n A, Schillaci R, Pardal E. Derivaci&oacute;n de LCR lumbo-peritoneal. Rev Arg Neurocirug&iacute;a 1985; 2, 1: 152-157.</li>
<li>Savolainen S, Paljarvi L, Vapalahti M. Prevalence of Alzheimer&rsquo;s disease in patients investigated for presumed normal pressure hydrocephalus: a clinical and neuropathological study. Acta Neurochir (Wien) 1999; 141 (8): 849-53.</li>
<li>Sellal F, Becker H. Potentially reversible dementia. Presse Med 2007; 36 (2Pt2): 289-98.</li>
<li>Tisell M, Hellstrom P, Ahl-Borjesson G, et al. Long-term outcome in 109 adult patients operated on for hydrocepahalus. Br J Neurosurg 2006; 20 (4): 214-21.</li>
</ol>]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/1021">
    <dcterms:title><![CDATA[Síndrome de Papiledema Aracnoidocele Intraselar]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[Entre la patología neuroftalmológica de dificil diagnóstico hemos encontrado en seis casos la asociación de aracnoidocele intraselar con papiledema.<br />
Se trata de 5 mujeres en la edad media de la vida, obesas, multíparas, y 1 hombre de 43 años, todos tenían antecedentes de cefalea de larga data y disminución de la agudeza visual.<br />
Los estudios neuro-radiológicos demostraron agrandamiento de la silla turca con inexistencia de patología tumoral o vascular de la región selar: en las NCG se apreció la entrada de aire en la silla turca y las mediciones de presión de L.R.C. (PL. Tornillo subdural) evidenciaron cifras por encima de la normalidad.<br />
Consideramos en base a la clínica y los estudios complementarios de diagnóstico que estos casos correspondían a un Síndrome de Hipertensión Endocraneana Benigna de larga data con herniación de la cisterna a través del diafragma selar, en consecuencia no hay indicación para realizar la corrección quirúrgica del aracnoidocele intraselar que no es la causa sino la consecuencia de la presión endocraneana elevada, al igual que el papiledema.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[A. Carrizo]]></dcterms:creator>
    <dcterms:creator><![CDATA[A. Kreutel]]></dcterms:creator>
    <dcterms:creator><![CDATA[S. Berner]]></dcterms:creator>
    <dcterms:creator><![CDATA[M. Acuña]]></dcterms:creator>
    <dcterms:creator><![CDATA[C. Yampolsky]]></dcterms:creator>
    <dcterms:creator><![CDATA[A. Basso]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 1990]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/1151">
    <dcterms:title><![CDATA[Síndrome de Ramsa y Hunt Postherpético. Manejo Neuroquirúrgico]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:creator><![CDATA[Schvarcz, J. R.]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Hugo N. Usarralde]]></dcterms:publisher>
    <dcterms:date><![CDATA[Agosto 1984]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/317">
    <dcterms:title><![CDATA[Síndrome de Tolosa-Hunt]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objetivo. Reportar 2 casos de pacientes con diagnóstico de síndrome de Tolosa-Hunt y realizar una revisión de la literatura de esta infrecuente patología.<br />
Descripción. Caso 1: Paciente femenina de 21 años de edad que consulto por oftalmoplejía dolorosa de 72 hs de evolución sin compromiso visual. Se realizó IRM de encéfalo que evidenció el engrosamiento difuso con realce postcontraste del seno cavernoso izquierdo. Se interpretó el cuadro como un STH. Caso 2: Paciente masculino de 45 años de edad que consultó por oftalmoplejía dolorosa de 2 meses de evolución y amaurosis del ojo derecho. Se le solicitó IRM de encéfalo que evidenció el engrosamiento difuso del seno cavernoso derecho. La retinografía evidenció la atrofia de la papila de dicho ojo. El cuadro se interpretó como un STH asociado a una neuritis óptica homolateral.<br />
Intervención. Caso 1. Inicio tratamiento con corticoides y carbemazepina lográndose a los 3 meses recuperación total sin recaídas posteriores. Caso 2. Se inicio tratamiento con corticoides. Actualmente se encuentra en control por consultorios presentando mejoría significativa de los síntomas sin mejoría de la amaurosis de ingreso.<br />
Conclusión. El STH es una entidad poco frecuente, con un probable origen autoinmunitario. Existen diversas causas que pueden simular este síndrome. Es mandatorio el análisis exhaustivo de estos pacientes, a fin de descartar otras patologías.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Diego F. Martínez]]></dcterms:creator>
    <dcterms:creator><![CDATA[ Juan Pablo Casasco]]></dcterms:creator>
    <dcterms:creator><![CDATA[Natalia Pendre]]></dcterms:creator>
    <dcterms:creator><![CDATA[Cristian De Bonis]]></dcterms:creator>
    <dcterms:creator><![CDATA[Silvia I. Berner]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Graciela Zúccaro]]></dcterms:publisher>
    <dcterms:date><![CDATA[Julio 2010]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/449">
    <dcterms:title><![CDATA[Síndrome del área motora suplementaria postoperatoria. Reporte de un caso]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Trabajos Breves]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objective. To analyze the anatomical and semiological features of the syndrome of the SMA throgh the presentation of a patient.<br />
Description. 55, female. Two yrs. history of generalized convultions. Left frontomedial lesion biopsied outwards. PA: low grade astrocytoma. Medically trated until feb. 2007. Sudden headache and right sided hemiparesis with crural predominance. Hoffmann and slight right hyper reflexia. MRI: expansion of primary lesion with intra lesional hemorrhage. Surgical resection. PA: mixed anaplastic glioma. The inmediate posoperative status was of a right CB plejia with severe apathy and abulia with right spatial hemineglect and hypofluent and anomic languaje, with preserved repetition and comprehension. Total recovery of paresis and languaje except when stressed.<br />
Discussion. The SMA is limited by cingulate cortex inferiorly, PMC posteriorly, and vértex superiorly. The anterior border is not well defined. SMA activates before the PMC, during planification and iniciation of movement. Is connected with the PMC, spinal cord neurons, basal ganglia and contralateral SMA. By intraoperative stimulation it could be established the somatotopic organization of SMA, with the the hindlimb posterior the forelimb intermediate and face anterior. Before the facial representation is the language area in the dominant hemisphere. Epileptogenic crisis originated in this area produce abduction and supination of the contralateral forelimb with deviation of the head to the same side. The syndrome of lesion of SMA includes: Global akinesis, proximal predominance of motor déficit, with secuelar difficulty for the fine movements of hands an a quick recuperation (days to weeks).<br />
Conclussion. The síndrome of the supplementary motor area is well recognized after medial frontal lobe surgery. Its features include reduction of spontaneous movements in the contralateral limbs and speech deficit. The impairment of volitional movements is the main finding. Recovery is complete in a short period of time.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Andrés Cervio]]></dcterms:creator>
    <dcterms:creator><![CDATA[Mario Espeche]]></dcterms:creator>
    <dcterms:creator><![CDATA[Rubén Mormandi]]></dcterms:creator>
    <dcterms:creator><![CDATA[Santiago Condomí Alcorta]]></dcterms:creator>
    <dcterms:creator><![CDATA[Jorge Salvat]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Horacio J. Fontana]]></dcterms:publisher>
    <dcterms:date><![CDATA[Septiembre 2007]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/811">
    <dcterms:title><![CDATA[Síndrome del Niño Sacudido<br />
Presentación de un Caso y Revisión de la Literatura]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Reporte de Caso]]></dcterms:description>
    <dcterms:creator><![CDATA[Gustavo Tróccoli]]></dcterms:creator>
    <dcterms:creator><![CDATA[Ernesto Alda]]></dcterms:creator>
    <dcterms:creator><![CDATA[Eugenio D&#039;Annuncio]]></dcterms:creator>
    <dcterms:creator><![CDATA[Antonio Giuliano]]></dcterms:creator>
    <dcterms:creator><![CDATA[Osvaldo Gutiérrez]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Julio C. Suárez]]></dcterms:publisher>
    <dcterms:date><![CDATA[Diciembre 1998]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/486">
    <dcterms:title><![CDATA[Síndrome del outlet torácico: ¿una patología siempre quirúrgica? Análisis de una serie de 31 cirugías realizadas por vía supraclavicular]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objetivo. El objetívo de este trabajo es analizar los resultados obtenidos en una serie de cirugías realizadas en esta patología. Material y métodos. Se analizaron todos los casos de cirugías de nervios efectuadas en el período 2003-2012, separando los casos con diagnóstico de outlet torácico operados con un período de seguimiento postoperatorio mínimo de 6 meses. Se buscaron los siguientes datos: edad, sexo, presencía de síntomas sensitivos y/ o motores, tipo de outlet (verdadero o disputado), resultado de los estudios neurofisiológicos y de imágenes, resultado de la cirugía, complicaciones postoperatorias y recidivas.<br />
Resultados. Se incluyeron 31 cirugías realizadas en 30 pacientes, 9 con diagnóstico de OW (8 mujeres) con un promedio de edad fue de 24,3 años, y 21 con OTD (18 mujeres) de 37,4 años en promedio. Un 90% de todos los casos de outlet presentaron alteraciones neurofisiológicas preoperatorias, y los estudios imagenológicos fueron anormales en 66,6%. Una vez realizada la exploración, el 100% de los OTV presentó una alteración anatómica claramente relacionada con la sintomatología, hecho observado sólo en el 36,7% de los OTD operados. El 87,5% de los OW mejoraron sus síntomas sensítivos luego de la cirugía, mientras que 77,7% mejoraron desde el punto de vista motor. Por el contrario, 45.4% de los OTD mejoraron permanentemente, 36,3% no tuvieron cambios, 13,6% mejoraron transitoriamente y 4,5% (un caso) empeoró. Las complicaciones postoperatorías fueron más frecuentes aunque transitorias en el grupo de OTV (3 casos sobre 9 operados, 33.3%) que en los OTD (3 casos sobre 22, un 13,6%).<br />
Conclusión. El OTV es una patología infrecuente cuyo tratamiento quirúrgico suele evolucionar favorablemente. En cambio, el OTD constituye un diagnóstico de exclusion y su tratamiento quirúrgico da un resultado bueno aunque inferior al del OTV. Un análisis reciente de la literatura indica que no existe evidencia estadísticamente concluyente sobre los criterios diagnósticos y terapéuticos de los pacientes que sufren estos síndromes.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Mariano Socolovsky]]></dcterms:creator>
    <dcterms:creator><![CDATA[Gilda Di Masi]]></dcterms:creator>
    <dcterms:creator><![CDATA[Daniela Binaghi]]></dcterms:creator>
    <dcterms:creator><![CDATA[Álvaro Campero]]></dcterms:creator>
    <dcterms:creator><![CDATA[Miguel Dominguez Páez]]></dcterms:creator>
    <dcterms:creator><![CDATA[Alberto Dubrovsky]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Marcelo Platas]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 2013]]></dcterms:date>
    <dcterms:dateAccepted><![CDATA[marzo 2013]]></dcterms:dateAccepted>
    <dcterms:dateSubmitted><![CDATA[febrero 2013]]></dcterms:dateSubmitted>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:language><![CDATA[Inglés]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[<ol>
<li>Peet RM, Henriksen JD, Anderson TP, Martin GM. Thoracic outlet syndrome: evaluation of a therapeutic exercise program. Proceedings of the Staff Meetings. Mayo Clinic 1956; 31(9): 281-7.</li>
<li>Gilliatt RW, Le Quesne PM, Logue V, Sumner AJ. Wasting of the hand associated with a cervical rib or band. J Neurol Neurosurg Psychiatry. 1970; 33(5): 615-24.</li>
<li>Redenbach DM, Nelems B. A comparative study of structures comprising the thoracic outlet in 250 human cadavers and 72 surgical cases of thoracic outlet syndrome. Eur J Cardiothorac Surg. 1998; 13(4): 353-60.</li>
<li>Povlsen B, Belzberg A, Hansson T, Dorsi M. Treatment for thoracic outlet syndrome.Cochrane Database Syst Rev 2010 Jan 20 (1)</li>
<li>Edwards DP, Mulkern E, Raja AN, Barker P. Trans-axillary first rib excision for thoracic outlet syndrome. J Royal College of Surgeons of Edinburgh 1999; 44(6) 362-5.</li>
<li>Juvonen T, Satta J, Laitala P, Luukkonen K, Nissinen J. Anomalies at the thoracic outlet are frequently in the general population. American J Surgery 1995; 170(1): 33-7.</li>
<li>FechterJD, Kuscher SH. The thoracic outlet syndrome. Orthopedics 1993; 16(11): 1243-51.</li>
<li>Willbourn AJ. The thoracic outlet syndrome is overdiagnosed. Archives of Neurology 1990; 47(3): 328-30.</li>
<li>Sheth RN, Campbell JN. Surgical treatment of thoracic outlet syndrome: a randomized trial comparing two operations. Journal of Neurosurgery. Spine 2005; 3(5): 355-63.</li>
<li>Lindgren KA. Conservative treatment of thoracic outlet syndrome: a 2-year follow-up. Archives of Physical Medicine and Rehabilitation 1997; 78(4): 373-8.</li>
<li>Jordan SE, Ahn SS, Freischlag JA, Gelabert HA, Machleder HI. Selective botulinum chemodenervation of the scalene muscles for treatment of neurogenic thoracic outlet syndrome. Annals of Vascular Surgery 2000; 14(4): 365-9.</li>
<li>Bhattacharya V, Hansrani M, Wyatt MG, Lambert D, Jones NAG. Outcome following surgery for thoracic outlet syndrome. European J of Vascular and Endovascular Surgery 2003; 26(2): 170-5.</li>
<li>Landry GJ, Moneta GL, Taylor LM Jr, EdwardsJM, PorterJM. Long-term functional outcome of neurogenic thoracic outlet syndrome in surgically and conservatively treated patient. J vasc surg 2001; 33:312-7.</li>
<li>Martens V, Bugden C. Thoracic outlet syndrome: a review of 67 cases. Canadian Journal of Surgery 1980; 23(4): 357-8.</li>
<li>Sallstróm J, Gjores JE. Surgical treatment of the thoracic outlet syndrome. Acta Chirurgica Scandinavica 1983; 149 (6): 555-60.</li>
</ol>]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/1012">
    <dcterms:title><![CDATA[Síndrome del Tunel Carpiano. Análisis de 90 Intervenciones.<br />
(Con sugerencias para evitar complicaciones)]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[El tratamiento del síndrome del túnel carpiano es simple pero puede estar asociado a complicaciones severas como la pérdida de oposición del pulgar y la formación de neuromas. causalgia, cicatrices hipertróficas y rigidez articular. Se analizan 90 descompresiones del nervio mediano realizadas en 80 pacientes con la misma técnica durante el periodo 1977-1987. En todos los pacientes el diagnóstico se confirmó mediante los estudios de conducción nerviosa para evitar cirugías innecesarias. La incisión se trazó medial al pliegue tenar y se extendió hasta el pliegue flexor del carpo con una inclinación de 45° para no dañar las ramas palmar cutánea y tenariana del mediano y evitar una cicatriz hipertrófica. No se efectuaron infiltraciones ni neurolisis del mediano para no producir una causalgia. La inmovilización postoperatoria no se prolongó más de 72 horas para evitar rigidez articular. No hubo mortalidad y las disestesias desaparecieron en el 97% de las descompresiones. Se discuten los detalles de la evaluación diagnóstica y de la técnica quirúrgica.<br />
Creemos que las complicaciones se pueden evitar con un buen conocimiento de la anatomía y teniendo en cuenta los cuidados técnicos ya mencionados.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[D. Gonzalez Moles]]></dcterms:creator>
    <dcterms:creator><![CDATA[J. J. M. Mezzadri]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Julio C. Suárez]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 1991]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/344">
    <dcterms:title><![CDATA[Sindromes de compresión neurovascular descompresión microvascular. Nuestra experiencia (2003-2009)]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Serie de Casos]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objective. To present and evaluate the surgical results of microvascular decompression in the treatment of trigeminal neuralgia, glossopharyngeal neuralgia and hemifacial spasm.<br />
Material and Method. We present 15 patients with compressive syndrome of cranial nerve during a period of 6 years: 11 patients with trigeminal neuralgia, 2 patients with glossopharyngeal neuralgia and 2 patients with hemifacial spasm.<br />
Results. We achieved total improvement in 8 patients and partial in 4. The most frequent symptom after surgery was hypoesthesia in V2 in patients with trigeminal neuralgia. 3 patients presented temporary facial paresis.<br />
Conclusion. Microvascular decompression is an effective treatment with low morbidity in patients with compressive syndrome of cranial nerves. The goal of treatment should be complete disappearance of symptoms.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Mariano Rinaldi]]></dcterms:creator>
    <dcterms:creator><![CDATA[Adrián Paris]]></dcterms:creator>
    <dcterms:creator><![CDATA[Matías Berra]]></dcterms:creator>
    <dcterms:creator><![CDATA[Heraldo Pares]]></dcterms:creator>
    <dcterms:creator><![CDATA[Ricardo Olocco]]></dcterms:creator>
    <dcterms:creator><![CDATA[ Francisco Papalini]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Graciela Zúccaro]]></dcterms:publisher>
    <dcterms:date><![CDATA[Julio 2009]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/760">
    <dcterms:title><![CDATA[Siringomielia y Tumores de Fosa Posterior]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:creator><![CDATA[D. H. D&#039;Osvaldo]]></dcterms:creator>
    <dcterms:creator><![CDATA[J. D. Oviedo]]></dcterms:creator>
    <dcterms:creator><![CDATA[J. M. Otero]]></dcterms:creator>
    <dcterms:creator><![CDATA[J. B. Mosconi]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Julio C. Suárez]]></dcterms:publisher>
    <dcterms:date><![CDATA[Diciembre 2000]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/943">
    <dcterms:title><![CDATA[Siringomielia: Actualizacion en Diagnóstico y Tratamiento]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Actualización]]></dcterms:description>
    <dcterms:abstract><![CDATA[La siringomielia es una cavitación tubular de la médula espinal independiente o dependiente del conducto ependimario cuya complejidad queda evidenciada por diferentes y disímiles clasificaciones.<br />
Su diagnóstico resulta dificultoso ante la baja incidencia de formas agudas y subagudas. La sintomatología dolorosa es la forma de presentación más frecuente (cefaleas, cervicalgias, dolores radiculares y cordonales) y también los trastornos de la marcha, sensitivos y tráficos.<br />
El examen neurológico puede evidenciar un síndrome lesional motor y / o sensitivo y otro sublesional (piramidal ,espinotalámico lateral).<br />
Los criterios neuropatológicos incluyen la diferenciación con la hidromielia; la relación con el canal ependimario, su comunicación con los espacios ventricular y subaracnoideo y las características de los bordes y del tejido medular que rodea la cavidad.<br />
La metodología diagnóstica incluye la tomografia axial computada (TAC) complementada con la mielografía y la resonancia magnética nuclear (IRM) que es el único estudio que permite apreciar la exacta extensión rostrocaudal de la cavidad.<br />
El tratamiento quirúrgico difiere según sea el caso de la siringomielia primaria o asociada a malformaciones y debe estar indicado ante la progresión del déficit neurológico. Existen diferentes técnicas quirúrgicas dependiendo de la patología y de la particular experiencia de los autores.<br />
El tratamiento de esta compleja enfermedad permite mejorar o estabilizar el cuadro clínico en un porcentaje significativo de pacientes.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Guillermo Larrarte]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Julio C. Suárez]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 1996]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/655">
    <dcterms:title><![CDATA[Sistemas de Trauma. Propuesta de Organización]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículos Varios]]></dcterms:description>
    <dcterms:abstract><![CDATA[El trauma constituye la principal causa de muerte para los grupos de edad de 1 a 45 años y debe ser considerada una enfermedad endémica, no solo en países desarrollados, sino también en países en vías de desarrollo. Sin embargo, el trauma es reconocido como &quot;la enfermedad negada de la sociedad moderna&quot;. Para ello, es necesario una aproximación sistemática para enfrentarla. Primero, se presenta una sucinta revisión de la experiencia legislativa y organizativa desarrollada en USA y Canadá en relación con la implementación de los sistemas y centros de trauma y la evidencia de su eficacia a través de diferentes métodos de evaluación como los paneles de expertos, los registros de trauma y los estudios basados en la población. Luego, se describe una propuesta organizativa de sistema de trauma emergencia y desastres para la Argentina. Esta propuesta está principalmente pensada sobre la base organizativa del municipio con niveles de desarrollo local, provincial, regional y nacional. Ese sistema debería estar preparado para enfrentar situaciones de trauma, emergencias y desastres no solo a nivel local sino también en caso de desastres de grandes proporciones. Se detallan definiciones de los diferentes niveles de atención del traumatizado y del entrenamiento del recurso humano. Debe adjudicarse una gran importancia a la implementación del registro nacional de trauma que permita al sistema conocer el impacto real de la enfermedad trauma y diseñar políticas de prevención apropiadas.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Jorge A. Neira]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Juan José Mezzadri]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Luis Augusto Lemme-Plaghos]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 2004]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/648">
    <dcterms:title><![CDATA[Sistemas de Trauma. Propuesta de Organización 2a. parte<br />
Proyecto de Regionalización a Nivel Nacional]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículos Varios]]></dcterms:description>
    <dcterms:creator><![CDATA[Dr. Jorge A. Neira]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Juan José Mezzadri]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Luis Augusto Lemme-Plaghos]]></dcterms:publisher>
    <dcterms:date><![CDATA[Julio 2004]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[1.	Neira J. Organización de la atención del traumatizado. Sistemas de Trauma. Centros de Trauma. En Prioridades en Trauma. San Román E, Neira J, Tisminetzky G (eds). Editorial Panamericana. Buenos Aires. 2002 pp 45-53.<br />
2.	Bosque L, Neira J. El término accidente. En Trauma Prioridades. San Román E, Neira J, Tisminetzky G (eds). Editorial Panamericana. Buenos Aires. 2002 pp 19-20.<br />
3.	Neira J, Bosque L, Zengotita S. Informe estadístico sobre trauma. Ario 2000. Sociedad Argentina de Medicina y Cirugía del Trauma y Red de trauma y Emergencia de la Secretaria de Salud del Gobierno de la Ciudad de Buenos Aires.<br />
4. Airey C, Franks A. Major trauma workload within a English Health Region. Injury 1995; 26: 25-31.<br />
5. Gorman D, Teanby D, Sinha M, Wotherspoon J, Boot DA, Molokhia A. The epidemiology of major injuries in Mersey Region and North Wales. Injury 1995; 26: 51-55.<br />
6.	Battistella F, Torabian S, Siadatan K. Hospital readmission after trauma: An analysis of outpatient complications. J Trauma 1997; 42: 10121016.<br />
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326. Rogers F, Reath D, Spain D. A prospective study of predictors or disability at 3 month after non-central nervous system trauma. J Trauma 1998; 44: 642-643.<br />
327. Sicignano A, Giudici D. Probability model of hospital death for severe trauma patients based on the simplified acule physiology score I: Development and validation. J Trauma 1997; 43: 585589.<br />
328. Richmond T, Kauder D, Schwab W. A prospective study of predictors or disability at 3 month after non-central nervous system trauma. J Trauma 1998; 44: 635-642.]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/715">
    <dcterms:title><![CDATA[Sobre comentarios críticos y...]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Editorial]]></dcterms:description>
    <dcterms:creator><![CDATA[Juan J. Mezzadri <br />
]]></dcterms:creator>
    <dcterms:creator><![CDATA[Luis Lemme-Plaghos ]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Juan José Mezzadri]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Luis Augusto Lemme-Plaghos]]></dcterms:publisher>
    <dcterms:date><![CDATA[Junio 2003]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/835">
    <dcterms:title><![CDATA[Subependimoma de Tercer Ventrículo]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Trabajos Breves]]></dcterms:description>
    <dcterms:creator><![CDATA[J. Mercuri]]></dcterms:creator>
    <dcterms:creator><![CDATA[E. Lis ]]></dcterms:creator>
    <dcterms:creator><![CDATA[W. Saborido]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Julio C. Suárez]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 1998]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/285">
    <dcterms:title><![CDATA[Subependimoma Sintomático del Piso del IV Ventrículo<br />
Reporte de un Caso y Revisión de la Literatura]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Revisión de la Literatura]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objetivo. Reportar un caso de esta infrecuente patología y realizar una revisión bibliográfica, analizando sus características clínicas y morbimortalidad.<br />
Descripción. Presentamos aquí el caso de un paciente de 64 años, el cual consulta por un síndrome vertiginoso de un año de evolución. En la IRM se observaba una lesión ocupante de espacio en el cuarto ventrículo, la cual media 5.3 x 4.1 x 4.2 cm. No se observaba franca hidrocefalia a pesar del tamaño de la misma.<br />
Intervención. Se efectuó una craniectomía suboccipital con laminectomía Cl. El tumor nacíafrancamente del piso del IV ventrículo. Se resecó la lesión enforma completa, efectuando cavitación y disección roma entre la cápsula y el piso del IV ventrículo. El paciente tuvo un postoperatorio tórpido dada la paresia de pares bajos que presentó durante las primeras semanas, por lo que fue intubado, traqueostomizado y alimentado por sonda nasoyeyunal. Al 5t0 mes de postoperatorio el paciente se hallaba independiente para sus actividades de la vida diaria. La resonancia postoperatoria evidenció la exéresis completa de la lesión.<br />
Conclusión. Los subependimomas que nacen del piso del IV ventrículo tienen una elevada morbimortalidad documentada. Sólo la minuciosa disección con microscopio y cuidados intensivos postoperatorios pueden mejorar este pronóstico.<br />
Palabras clave: ependimoma, subependimoma, tumor del IV ventrículo.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Ignacio J. Barrenechea]]></dcterms:creator>
    <dcterms:creator><![CDATA[Néstor Yunis]]></dcterms:creator>
    <dcterms:creator><![CDATA[Daniel Rovira]]></dcterms:creator>
    <dcterms:creator><![CDATA[José Carlos Nallino]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Rafael Torino]]></dcterms:publisher>
    <dcterms:date><![CDATA[2011]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/520">
    <dcterms:title><![CDATA[Subluxación atlanto-axial anterior secundaria a osteoartritis: reporte de un caso]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Reporte de Caso]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objetivo. Describir un caso de subluxación atlantoaxial anterior secundaria a una osteoartritis.<br />
Descripción. Un paciente masculino de 60 años de edad con una tetraparesia moderada, rápidamente progresiva, piramidalismo y trastornos sensitivos superficiales y profundos, de cuatro meses de evolución. Las radiografías de columna cervical frente y perfil muestraron una subluxación atlanto-axial anterior. La resonancia magnética mostró un tejido patológico periodontoideo con extensión a articulares y mielomalacia compresiva a nivel C1-C2 a expensas de elementos posteriores por la subluxación. Las radiografías del resto de la columna, manos, rodillas y hombros mostraron cambios degenerativos artrósicos. Se realiza una interconsulta con el servicio de reumatología que, luego del analizar los resultados serológicos, clínicos y radiológicos, arribó al diagnóstico de osteoartritis.<br />
Intervención. Consistió en un abordaje posterior, con apertura del foramen magno, laminectomía de C1 y osteosíntesis occipitocervical con la colocación de injertos óseos autólogos.<br />
Conclusión. La osteoartritis es una causa infrecuente de subluxación atlantoaxial anterior. Más allá de la patología causal, sabemos que el tratamiento quirúrgico es fundamental en los pacientes sintomáticos.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Paula Ferrara]]></dcterms:creator>
    <dcterms:creator><![CDATA[Natalia Spaho]]></dcterms:creator>
    <dcterms:creator><![CDATA[Eduardo Vecchi]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Horacio J. Fontana]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Juan José Mezzadri]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 2006]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/841">
    <dcterms:title><![CDATA[Subluxación Atloideo Axoidea Tratamiento Quirúrgico. Abordaje Transoral ]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Trabajos Breves]]></dcterms:description>
    <dcterms:creator><![CDATA[A. L. Gidekel]]></dcterms:creator>
    <dcterms:creator><![CDATA[A. Cristini]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Julio C. Suárez]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 1998]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/1315">
    <dcterms:title><![CDATA[Subluxación cervical espondiloartrósica]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Trabajos Breves]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objective: To analyze the biomechanical and anatomical factors and the surgical treatment and outcome in cervical degenerative spondylolisthesis.<br />
Method: Between 1978 and 2003, 223 patients underwent surgery for cervical spondylotic myelopathy. 41 patients presented with degenerative subaxial subluxation. According to the Nurick scale, 26 were grade 2, 14 were grade 3 and 1 was grade 4. Those with degenerative subluxation were older compared to those who liad only spondylotic myelopathy (63 vs. 13 years). Theformer also corresponded to higher grades of the Nurick scale. All patients underwent static and dynamic plain radiographs, and MRI. We found 29 subluxations of one level, 11 patients wíth affection of two levels and 1 with three levels. 33 patients liad unstable subluxations. The most commonly affected levels were C3-C4 and C4-05. In all the cases, an anterior approach with autogenous bone graft and locking plate was performed.<br />
Results: After surgical treatment, 21 grade 2 patients improved one grade, while 5 improved two grades; 9 grade 3 patients improved one grade and 1 two grades; the grade 4 patient improved one grade.<br />
Conclusion: Cervical degenerative subluxation is seen in older patients, usually associated with severe myelopathy and spondylotic changes. The cervical instability is more common in the C3-C4 and C4-05 segments. In oil the cases of our group there was a loss of cervical lordosis or kyphosis with ankylosis of the lower cervical spine. The anterior approach with autogenous bone graft and locking Mate, allowed spinal cord decompression and cervical fusion.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Jorge Shilton]]></dcterms:creator>
    <dcterms:creator><![CDATA[Nilda Ester Goldenberg]]></dcterms:creator>
    <dcterms:creator><![CDATA[ José Carlos Morales]]></dcterms:creator>
    <dcterms:creator><![CDATA[Alberto Zilio]]></dcterms:creator>
    <dcterms:creator><![CDATA[Lucrecia Valdez]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Luis Augusto Lemme-Plaghos]]></dcterms:publisher>
    <dcterms:date><![CDATA[Octubre 2003]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[Perlman R, Hawes LE: Cervical spondylolisthesis. J Bone Joint Surg 1951; 33A: 1012-3.<br />
<br />
Pellengahr C, Pfahler M, Kuhr et al: Influence of facet joint angles and asymmetric disc collapse on degenerative olisthesis of the cervical spine. Orthopedics 2000; 23: 697-701<br />
<br />
Boulos AS, Lovely TJ: Degenerative cervical spondylolisthesis: Diagnosis and management in five cases. J Spinal Disord 1996; 9: 241-5.]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/71">
    <dcterms:title><![CDATA[Subtalamotomía bilateral diferida guiada por microrregistros. Resultados motores a un año]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[<p><strong>RESUMEN</strong><br /> <strong>Introducci&oacute;n:</strong> Diferentes t&eacute;cnicas quir&uacute;rgicas representan una alternativa para el tratamiento de la enfermedad de Parkinson avanzada (EP). El gran desarrollo en las &uacute;ltimas d&eacute;cadas de modernas t&eacute;cnicas de im&aacute;genes sumado a los avances del registro neurofisiol&oacute;gico nos permiten localizar y generar lesiones en forma precisa, con bajo n&uacute;mero de complicaciones y excelentes resultados funcionales.<br /> <strong>Objetivo:</strong> Describir los resultados motores obtenidos a los 12 meses post-operatorios de pacientes con diagn&oacute;stico de EP avanzada sometidos a subtalamotom&iacute;a bilateral diferida guiada por microrregistro.<br /> <strong>Material y M&eacute;todos:</strong> Se seleccionaron 9 pacientes con diagn&oacute;stico de EP avanzada de acuerdo a los criterios de la United Kingdom Parkinson&acute;s Disease Brain Bank, evaluando los efectos de la subtalamotom&iacute;a bilateral en dos tiempos quir&uacute;rgicos.<br /> <strong>Resultados:</strong> Una marcada mejor&iacute;a de los s&iacute;ntomas cardinales como rigidez, bradicinesia y temblor se observ&oacute; en todos los pacientes, fundamentalmente en la condici&oacute;n de &ldquo;off&rdquo;, y en menor medida tambi&eacute;n en condici&oacute;n &ldquo;on&rdquo;. Se destaca un significativo control de las fluctuaciones motoras y discinesias.<br /> <strong>Conclusi&oacute;n:</strong> La subtalamotom&iacute;a bilateral diferida guiada por microregistro es un m&eacute;todo &uacute;til y efectivo en el tratamiento de los s&iacute;ntomas motores de la Enfermedad de Parkinson.</p>
<p><strong>Palabras Clave:</strong> Enfermedad de Parkinson; Subtalamotom&iacute;a; Microrregistro</p>
<p><strong>ABSTRACT</strong><br /> <strong>Introduction:</strong> In the treatment of advanced Parkinson&acute;s disease (PD) there are different surgical techniques that represent a valid alternative. The development of modern images techniques and neurofisiological recording allow us to generate accurate lesions, with low complications and excellent functional results.<br /> <strong>Objective:</strong> Describe motor results 12 months post-surgical of patients with advanced PD submitted to bilateral and deferred subthalamotomy guided by microrecording. <br /> <strong>Material and Methods: </strong>We selected 9 patients with advanced PD diagnosis in concordance with United Kingdom Parkinson`s Disease Brain Bank criteria, evaluating the effects of bilateral subthalamotomy in two different surgical steps.<br /> <strong>Results:</strong> A marked improvement of cardinal symptoms such as rigidity, bradykinesia and tremor was observed in all patients, fundamentally in &ldquo;off&rdquo; condition, and lesser extent in &ldquo;on&rdquo; condition. We highlighted a significant control of motor fluctuations and dyskinesias.<br /> <strong>Conclusion:</strong> Bilateral and deferred suthalamotomy guided by microrecording is an effective and useful method in treatment of cardinal motor symptoms of PD.</p>
<p><strong>Key words:</strong> Parkinson`s Disease; Subthalamotomy; Microrecording</p>]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Sergio Pampin]]></dcterms:creator>
    <dcterms:creator><![CDATA[Santiago L. Driollet]]></dcterms:creator>
    <dcterms:creator><![CDATA[Fernando Leiguarda]]></dcterms:creator>
    <dcterms:creator><![CDATA[Javier Ziliani]]></dcterms:creator>
    <dcterms:creator><![CDATA[Gastón Bartoli]]></dcterms:creator>
    <dcterms:creator><![CDATA[Juan PabloTravi]]></dcterms:creator>
    <dcterms:creator><![CDATA[Darío Adamec]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Marcelo Platas]]></dcterms:publisher>
    <dcterms:date><![CDATA[Diciembre 2014]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[<p><strong>BIBLIOGRAF&Iacute;A</strong></p>
<ol>
<li>Hughes AJ, Daniel SE, Blankson S. A Clinicopathological study of 100 cases of Parkinson&acute;s disease. J Neurol Neurosurg Psychiatry 1992; 55:181-4.</li>
<li>Goetz CG, Nutt JG, Stebbins GT. The Unified Dyskinesia Rating Scale: presentation and clinimetric profile. Mov Disord. 2008 Dec 15;23(16):2398-403.</li>
<li>Sergio Pampin, Santiago Driollet Laspiur, Fernando Leiguarda, Juan Pablo Travi, Dario Adamec. Subtalamotom&iacute;a por Radiofrecuencia: Aspectos T&eacute;cnico-Quir&uacute;rgicos. REV ARGENT NEUROC VOL. 27, N&ordm; 2: 67-71. 2013.</li>
<li>Jourdain VA, Schechtmann G, Di Paolo T. Subthalamotomy in the treatment of Parkinson's disease: clinical aspects and mechanisms of action. J Neurosurg. 2014 Jan; 120 (1): 140-51.</li>
<li>Deligny C, Drapier S, Verin M, Lajat Y, Raoul S, Damier P. Bilateral subthalamotomy through DBS electrodes: a rescue option for device-related infection. Neurology 73: 1243-1244. 2009.</li>
<li>Hirashima Y, Ikeda H, Asahi T, Shibata T, Noguchi K, Shima F, et al. Mechanical injury of the subthalamic area during stereotactic surgery followed by improvement of trunk, neck, and face tremor- case report. Neurol Med Chir (Tokyo) 45:484-486, 2005.</li>
<li>Keep MF, Mastrofrancesco L, Erdman D, Murphy B, Ashby LS. Gamma Knife subthalamotomy for Parkinson disease: the subthalamic nucleus as a new radiosurgical target. Case report. J Neurosurgery 97 (5 suppl): 592-599, 2002.</li>
<li>Obeso JA, Jahanshashi M. Alvarez L, Macias R. Pedroso I, Wilkinson L, et al. What can man do without basal ganglia motor output? The effect of combined unilateral subthalamotomy and pallidotomy in a patient with Parkinson&acute;s desease. Exp Neurol 220: 283-292, 2009.</li>
<li>Alvarez L, Macias R, Lopez G, Alvarez E, Pavon N, Rodriguez-Oroz M.C, Juncos J, Maragoto C, Guridi J, Litvan I, Tolosa E, Koller W, Vitek J, DeLong MR, Obeso J.A. Bilateral subthalamotomy in Parkinson&rsquo;s disease:initial and long-term response. Brain (2005), 128, 570&ndash;583.</li>
<li>Tseng HM, Su PC, Liu HM, Liou HH, Yen RF. Bilateral subthalamotomy for advanced Parkinson disease. Surg Neurol. 2007;68 Suppl 1:S43-50; discussion S50-1.</li>
<li>Su PC, Tseng HM, Liu HM, Yen RF, Liou HH. Subthalamotomy for advanced Parkinson disease. J. Neurosurgery 2001/2, 67: 598-606.</li>
<li>Vilela F, da Silva DJ. Unilateral subhtalamic nucleus lesioning: a safe and effective treatment for</li>
<li>Parkinson&rsquo;s disease. Arq Neuropsiquiatr. 2002; 60: 935&ndash;48.<br /> Patel NK, Heywood P, O&rsquo;Sullivan K, McCarter R, Love S, Gill SS. Unilateral subthalamotomy in the treatment of Parkinson&rsquo;s disease. Brain 2003; 126: 1136&ndash;45.</li>
<li>Tseng HM, Su PC, Liu HM, Liou HH, Yen RF. Bilateral subthalamotomy for advanced Parkinson disease. Surg Neurol. 2007;68 Suppl 1:S43-50; discussion S50-1.</li>
<li>Alvarez L, Macias R, Pavon N, Lopez G, Rodriguez-Oroz MC, Rodriguez R, et al. Therapeutic efficacy of unilateral subthalamotomy in Parkinson&acute;s disease: results in 89 patients followed for up to 36 months. J Neurol Neurosurg Psychiatry 80:979-985, 2009.</li>
<li>Merello M, Tenca E, P&eacute;rez Lloret S, Mart&iacute;n ME, Bruno V, Cavanagh S, Antico J, Cerquetti D, Leiguarda R. Prospective randomized 1-year follow-up comparison of bilateral subthalamotomy versus bilateral subthalamic stimulation and the combination of both in Parkinson's disease patients: a pilot study.Br J Neurosurg. 2008 Jun;22(3):415-22.</li>
<li>Krack P, Limousin P, Benabid AL, Pollak P. Chronic stimulation of subthalamic nucleus improves levodopa-induced dyskinesias in Parkinson&acute;s disease. Lancet 350: 1676, 1997.</li>
<li>Obeso JA, Rodriguez MC, Guridi J, Alvarez L, Alvarez E, Macias R, et al. Lesion of the basal ganglia and surgery for Parkinson disease. Arch Neurol 2001b; 58: 1165&ndash;6.</li>
<li>Alvarez L, Macias R, Guridi J, Lopez G, Alvarez E, Maragoto CT, et al. Dorsal subthalamotomy for Parkinson&rsquo;s disease. Mov Disord. 2001; 16: 72&ndash;8.</li>
<li>Rodriguez MC, Guridi OJ, Alvarez L, Mewes K, Macias R, Vitek J, DeLong MR, Obeso JA. The subthalamic nucleus and tremor in Parkinson's disease. Mov Disord. 1998;13 Suppl 3:111-8.</li>
<li>Gill SS, Heywood P .Bilateral dorsolateral subthalamotomy for advanced Parkinson&rsquo;s disease. The Lancet. Vol 350. Octubre 25, 1997.</li>
<li>Molinuevo JL, Valldeoriola F, Tolosa E, Rumia` J, Valls-Sole&acute; J, Rolda&acute;n H, Ferrer E. Levodopa withdrawal after bilateral subthalamic nucleus stimulation in advanced Parkinson disease. Arch Neurol 2000; 57: 983&ndash;8.</li>
<li>Lozano AM. The subthalamic nucleus: myth and opportunities. Mov. Disord 2001; 16: 183-4.</li>
<li>Vilela Filho O, Silva DJ, Souza HA, Cavalcante JE, Sousa JT, Ferraz FP, Silva LG, Santos LF. Stereotactic subthalamic nucleus lesioning for the treatment of Parkinson's disease. Stereotact Funct Neurosurg. 2001;77(1-4):79-86.</li>
<li>Chen CC, Lee ST, Wu T, Chen CJ, Huang CC, Lu CS. Mov Disord. Hemiballism after subthalamotomy in patients with Parkinson's disease: report of 2 cases. 2002 Nov; 17(6):1367-71.</li>
<li>Doshi P, Bhatt M. Hemiballism during subthalamic nucleus lesioning. Mov Disord 17:848-849, 2002.</li>
<li>Benabid AL, Benazzouz A, Limousin P, Koudsie A, Krack P, Piallat B, Pollak P. Dyskinesias and the subthalamic nucleus. Ann Neurol. 2000 Apr;47 (4 Suppl 1):S189-92.</li>
<li>Guridi J, Obeso JA. The subthalamic nucleus, hemiballismus and Parkinson's disease: reappraisal of a neurosurgical dogma. Brain. 2001 Jan;124(Pt 1):5-19.</li>
<li>Bickel S, Alvarez L, Macias R, Pavon N, Leon M, Fernandez C, Houghton DJ, Salazar S, Rodr&iacute;guez-Oroz MC, Juncos J, Guridi J, Delong M, Obeso JA, Litvan I. Cognitive and neuropsychiatric effects of subthalamotomy for Parkinson's disease. Parkinsonism Relat Disord. 2010 Sep;16(8):535-9.</li>
<li>Su PC, Tseng HM, Liou HH. Postural asymmetries following unilateral subthalomotomy for advanced Parkinson's disease. Mov Disord. 2002 Jan;17(1):191-4.</li>
</ol>]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/9">
    <dcterms:title><![CDATA[Subtalamotomía por Radiofrecuencia: Aspectos Técnico-Quirúrgicos]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Nota Técnica]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objetivo: Describir la técnica estereotáxica de subtalamotomía por radiofrecuencia utilizando microrregistro intraoperatorio con semimicroelectrodo.<br />
Descripción: Se detalla la técnica efectuada sobre 51 subtalamotomías desde el año 2011, en pacientes con diagnóstico de Enfermedad de Parkinson (EP) refractaria al tratamiento. La técnica quirúrgica consta de tres etapas: adquisición de las imágenes estereotácticas, localización anatómo-funcional de blanco quirúrgico y lesión por radiofrecuencia de la región sensitivo-motora del núcleo. Se describe el método utilizado para la exploración de la somatotopía del NST que permite la posterior lesión, confiriendo seguridad y efectividad al procedimiento. <br />
Conclusión: La subtalamotomía constituye una técnica segura y efectiva realizada en grupos entrenados con los recursos técnicos necesarios, para el tratamiento de la EP refractario, constituyendo una alternativa adaptable a nuestro medio con el objetivo mejorar la calidad de vida.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Sergio Pampin]]></dcterms:creator>
    <dcterms:creator><![CDATA[Santiago Driollet Laspiur]]></dcterms:creator>
    <dcterms:creator><![CDATA[Fernando Leiguarda]]></dcterms:creator>
    <dcterms:creator><![CDATA[Juan Pablo Travi]]></dcterms:creator>
    <dcterms:creator><![CDATA[Dario Adamec]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Marcelo Platas]]></dcterms:publisher>
    <dcterms:date><![CDATA[Junio 2013]]></dcterms:date>
    <dcterms:bibliographicCitation><![CDATA[Goetz CG, De Long MR, Penn RD, Bakay RA. Neurosurgical horizons in Parkinson's disease. Neurology. 1993 Jan;43(1):1-7.]]></dcterms:bibliographicCitation>
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    <dcterms:bibliographicCitation><![CDATA[Alvarez L, Macias R, Rodriguez-Oroz MC, LopezG, Alvarez E, Guridi J, Juncos J, Tolosa ES, KollerWC, Litvan I, Vitek J, De Long MR, Obeso JA. Bilateralsubthalamotomy in Parkinson´s disease. Neurology 2001; 56 (3): A273 (abstr).]]></dcterms:bibliographicCitation>
    <dcterms:bibliographicCitation><![CDATA[López Flores G, Fernández R, Macías R, Álvarez L, Teijeiro Amador J, Alvarez E, Maragoto C, Cruz O, Fermín E, Jordán J, Palmero R, Rodríguez R, Carballo-Barreda M. Subtalamotomía Estereotáctica. Nota Técnica. Revista CENIC Ciencias Biológicas 2004; 35 (2): 45-56.]]></dcterms:bibliographicCitation>
    <dcterms:bibliographicCitation><![CDATA[López Flores G, Miguel Morales J, Teijeiro Amador J, Fernández R, Álvarez L, Macías R, Cruz O, Fermín E, Palmero R, Rodríguez R, Carballo-Barreda M, Mosquera Betancourt G. Localización bilateral y simetría del núcleo subtalámico. Rev Neurol 2003; 37 (5): 404-412.x]]></dcterms:bibliographicCitation>
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</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/659">
    <dcterms:title><![CDATA[Surgical Management Of Cerebral Cavernous Malformations (Cavernomas) Causing Seizures In Children: The Cheo Experience. ]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[Purpose: To review seizure outcome after surgery for cerebral cavernous malformation (CCM) in children presenting with seizures as the main symptom.<br />
Material and Method: We retrospectively reviewed the cases of 10 patients with cerebral cavernous malformations presenting with seizures as the main symptom. These 10 patients were treated surgically between 1988 and 2004, at our institution. There were 5 males and 5 females, age range from 1 yr 8 months to 1 5 yrs (mean 8 yrs).The clinical histories, and radiological findings, patholog y and operative reports were reviewed. Imaging studies were done asfollows: CT + Angiogram in 4 patients, CT + MRI + MRA in 3 patients, CT + MRI in 2 patients.<br />
The cavernomas were located as follows: temporal lobe: 5 cases; parietal lobe: 3 cases (2 in sensory- motor strip); and frontal lobe: 2 cases. All patients had diagnostic EEG, and vine (90 %) were on antiepileptic drugs. Five patients had partial complex seizures and fine generalized tonic- clonic seizures. Seven (70%) patients underwent tailored complete lesionectorny and epileptic focus resection as determined by the use of intraoperative electrocorticography(ECOG). One patient (10%) underwent complete lesionectomy and partialfocus resection. Two patients (20%) with lesions in the sensory motor cortex underwent complete lesionectomy only. Mean follow up time was 5 yrs (range 3 months - 20 yrs).<br />
Results: All patients tolerated surgery well without morbidity or mortality. All patients are seizurefree. Seven (100%) patients who unclerwent tailored complete lesionectomy and epileptic focus resection are seizure free with no antiepileptic medication. One patient who underwent lesionectomy and partial focus resection is seizure free on rnedication. The two patients who underwent lesionectomy only, are both seizure free, one patient still requiring antiepileptic medication.<br />
Conclusion: For cerebral cavernous malformations presenting with seizures in this age group, complete microsurgical excision is the treatment of choice. Surgery should be preceded by careful electroclinical, neuroimaging, anatomical and functional evaluation. The good seizure outcome results (100%) after surgical intervention in our patients are probably related to both the lesionectomy and the excision of the epileptogenic focus in non eloquent areas (80%). Nevertheless, the two patients with lesions located in eloquent cortex that underwent complete lesionectomy abone (20%), are also seizurefree. Based on thesefindings it would appear that complete lesionectomy is the main variable in determining seizure outcorne after surgeryfor CCMs presenting with seizures. Larger series will be required to statistically support this assumption.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Enrique C.G. Ventureyra]]></dcterms:creator>
    <dcterms:creator><![CDATA[Munyao Nzau<br />
]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Edgardo Schijman]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Hugo Pomata]]></dcterms:publisher>
    <dcterms:date><![CDATA[Mayo 2004]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Inglés]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/955">
    <dcterms:title><![CDATA[Surgical Neuroangiography, vol.5]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Novedades Editoriales]]></dcterms:description>
    <dcterms:creator><![CDATA[Luis Lemme Plaghos]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Julio C. Suárez]]></dcterms:publisher>
    <dcterms:date><![CDATA[Septiembre 1995]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/239">
    <dcterms:title><![CDATA[Tablero de Gestión de Unidades Operativas de Neurocirugía]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[El presente trabajo tiene por objetivo exponer un modelo de tablero de gestión hospitalaria para su implementación específica en los Servicios de Neurocirugía, siendo su usuario el Jefe de Servicio. Se enmarca en la necesidad de optimizar elfuncionamiento de los Servicios de Neurocirugía a losfines de proveer una mejor gestión clínica y de pacientes, en la expectativa de obtener una mejoría de la eficiencia y calidad del trabajo neuroquirárgico en el seno de una institución. Se ha efectuado una revisión sistemática de las variables de uso en administración de salud y reconociendo entre ellos los indicadores que sirven para medir cambios (OMS). Se han seleccionado para la confección del Tablero de Gestión de Unidades Operativas de Neurocirugía (Servicios de Neurocirugía en la nomenclatura clásica) aquellos que presentan utilidad a los efectos de las incumbencias de la especialidad Neurocirugía. Se ha incorporado el concepto de evento centinela de la especialidad para su rápida detección y toma de curso de acción por la jefatura. ]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Javier Luis Gardella]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Rafael Torino]]></dcterms:publisher>
    <dcterms:dateAccepted><![CDATA[agosto de 2011]]></dcterms:dateAccepted>
    <dcterms:dateSubmitted><![CDATA[julio de 2011]]></dcterms:dateSubmitted>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:language><![CDATA[Inglés]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[<ol>
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<li>Kaplan R, Norton D. Cuadro de Mando Integral. ed. Gesti&oacute;n 2000.</li>
<li>Sampieri R, Fen&aacute;ndez Collado C, Lucio P. Metodolog&iacute;a de la Investigaci&oacute;n. Segunda edici&oacute;n. Mc Graw Hill: M&eacute;xico 1998.</li>
<li>Laguna Y, Arbeloa P, Remacha J, Lerchundi L. Larracoechea M. Calidad de la Codificaci&oacute;n de los Procedimientos Quir&uacute;rgicos y su Influencia en los Grupos Relacionados con el Diagn&oacute;stico.<strong> J Papeles M&eacute;dicos</strong> 2005; 15: 1-3.</li>
<li>Comisi&oacute;n Nacional de Clasificaci&oacute;n de Enfermedades (CNCE) Clasificaci&oacute;n de Procedimientos en Salud. Ministerio de Salud. . Direcci&oacute;n de Estad&iacute;sticas e Informaci&oacute;n de Salud. OPS. Bs. As. 2004.</li>
<li>L&oacute;pez Arbeloa P. Los GRD como sistema de medida del producto hospitalario. Biblioteca Basurto. Bilbao. 2004.</li>
<li>Mc Leod R. Sistemas de Informaci&oacute;n Gerencia'. Pearson Education. 2000.</li>
<li>Salvador Peir&oacute;. Evaluaci&oacute;n de calidad a partir del conjunto m&iacute;nimo de datos b&aacute;sicos (CMBD) al alta hospitalaria. www.svneurolog&iacute;a.org/ congreso accesada 10 mayo 2010.</li>
<li>Instituto Polit&eacute;cnico Nacional. Secretar&iacute;a T&eacute;cnica. Metodolog&iacute;a para el an&aacute;lisis FODA. M&eacute;xico. 2002.</li>
</ol>]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/802">
    <dcterms:title><![CDATA[Talamotomía Vim Vs. Palidotomía Pósteroventral como Alternativas en el Tratamiento de los Movimientos Anormales. <br />
Un estudio Multicéntrico de 474 Casos.<br />
]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[Se resume la experiencia en técnicas de Neurocirugía estereotáctica y funcional para el tratamiento de movimientos anormales del grupo de Neurocirugía estereotáctica del Hospital Universitario de Gunma, Japón, con los de nuestro grupo de trabajo, primero en La Habana y luego nuestra actual serie en el Centro de Restauración Neurológica en Buenos Aires. Fueron sometidos a Talamotomía Vim 431 pacientes (247 eran parkinsonianos temblorosos, 28 presentaban temblor intencional asociado a parálisis cerebral, 22 temblor postraumático, 18 temblor del escribiente, 93 temblor esencial y el resto otras patologías), mientras que 43 pacientes fueron sometidos a Palidotomía posteroventral. En todos los casos los procedimientosfueron realizados usando un marco estereotáctico de Leksell, guiado por planeamiento quirúrgico automatizado y microrregistro electrofisiológico profundo. Los resultados indican que la Talamotomía es una técnica quirúrgica segura, confiable y eficiente para el tratamiento de trastornos del movimiento predominantemente temblorosos, mientras que la Palidoto mía Selectiva Posteroventral lo es para tratar pacientes afectados con EP severa con rigidez, hipocinesia y diskinesias inducidas por L-Dopa como síntomas predominantes y es menos eficiente en pacientes donde predomina el temblor,]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Norberto Contreras]]></dcterms:creator>
    <dcterms:creator><![CDATA[Roberto Quiñones Molina]]></dcterms:creator>
    <dcterms:creator><![CDATA[Chihiro Ohyel]]></dcterms:creator>
    <dcterms:creator><![CDATA[Hilda Molina]]></dcterms:creator>
    <dcterms:creator><![CDATA[Ing. Ignacio Ortega]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Julio C. Suárez]]></dcterms:publisher>
    <dcterms:date><![CDATA[Diciembre 1998]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/1199">
    <dcterms:title><![CDATA[Técnica de libre flotación foraminal para discectomía endoscópica percutánea lumbar (PELD): nota técnica]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Nota Técnica]]></dcterms:description>
    <dcterms:abstract><![CDATA[RESUMEN<br />
La Discectomía Percutánea Endoscópica Lumbar es una técnica mínimamente invasiva para el tratamiento de las hernias discales lumbares con resultados comparables a la microdiscectomía lumbar. La principal complicación, los fragmentos discales remanentes, son en gran parte producidos por la limitación de movimiento del endoscopio una vez realizada la punción.<br />
Presentamos una nota técnica y aplicación en un caso ejemplo de lo que hemos llamado “libre flotación foraminal”: un acceso al espacio epidural por vía transforaminal que mantiene la libertad de movimiento y permite una exploración endoscópica en busca de fragmentos remanentes.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Facundo Van Isseldyk]]></dcterms:creator>
    <dcterms:creator><![CDATA[Julián Pastore]]></dcterms:creator>
    <dcterms:creator><![CDATA[Telmo Nicola]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Mariano Socolovsky]]></dcterms:publisher>
    <dcterms:date><![CDATA[Junio 2018]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[BIBLIOGRAFÍA<br />
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Lee SH, Kang HS, Choi G, Kong BJ, Ahn Y, Kim JS, Lee HY. Foraminoplastic ventral epidural approach for removal of extruded herniated fragment at the L5- S1 level. Neurol Med Chir (Tokyo) 2010; 50:1074-1078.  <br />
Ruetten S, Komp M, Merk H, Godolias G. Full endoscopic interlaminar and transforaminal lumbar discectomy versus conventional microsurgical technique: a prospective, randomized,  controlled study. Spine (Phila Pa 1976) 2008;33:931-9<br />
Ruetten S, Komp M, Merk H, Godolias G. Surgical treatment for lumbar lateral recess stenosis with the full-endoscopic interlaminar approach versus conventional microsurgical technique: A prospective, randomized, controlled study. J Neurosurg Spine 2009; 10:476-485]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/533">
    <dcterms:title><![CDATA[Técnicas de Enseñanza (&quot;Enseñar a Enseñar&quot;)]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículos Varios]]></dcterms:description>
    <dcterms:abstract><![CDATA[Es un error común pensar que la docencia es un &quot;arte intuitivo&quot;. Existen una serie de técnicas didácticas que pueden mejorar el rendimiento educativo. Es importante que el docente conozca las caracteristicas de sus estudiantes, prepare estrategias para captar la atención del auditorio y se adapte a sits alumnos. Debemos tener en cuenta que la ventana de atención de nuestro auditorio tiene un máximo de 60 minutos y que un adulto solo aprende los conocimientos que aplica. Si bien una presentación se divide tradicionalmente en tres secciones: introducción, desarrollo y conclusiones, el &quot;peso&quot; pedagógico de estas tres partes no es igual. El principio y el final son, desde un punto de vista de is motivation de su auditorio, los más importantes. La introducción debe ganar la atención del auditorio. establecer la relevancia del tema, plantear los objetivos de la clase y definir la estructura de la charla. Durante el desarrollo de la clase se deben explicar los conceptos. dar ejemplos y excepciones, repasarlos y preparar la transition para el proximo concepto. Ins conclusiones deben resumir los puntos principales del desarrollo, contestar las preguntas planteadas por los objetivos y estimular las preguntas del auditorio. Finalmente se debe exponer a la presentación a un &quot;control&quot; de calidad y de filtros de comunicación.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Roberto Rosler]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Horacio J. Fontana]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Juan José Mezzadri]]></dcterms:publisher>
    <dcterms:date><![CDATA[Diciembre 2005]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/480">
    <dcterms:title><![CDATA[Técnicas de resonancia magnética como herramientas esenciales para el abordaje de tumores del SNC]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objetivo. Mostrar la capacidad de las técnicas especiales de RMI (Espectroscopía , Difusión, Perfusión, ADC, Tractografía y RMI funcional (RMf) para: 1. Valorar las características intrínsecas (bioquímicas, grado de malignidad e infiltración) y extrínsecas (relación con las estructuras cerebrales normales) de las procesos expansivos del sistema nervioso central . 2. Decidir con la RMf la táctica quirúrgica más apropiada para cada caso.<br />
Material y método. Se presentan dos pacientes del sexo masculino de 35 y 40 años respectivamente, con lesiones tumorales en áreas elocuentes motoras del hemisferio cerebral derecho a los cuales se le efectuó cirugía, planificación y resección quirúrgica en base a los estudios de RMf (Philips 1,5 T Intera Achieva)<br />
Resultados. La localización de las áreas elocuentes mediante RMF y los hallazgos basados en las técnicas imagenológicas descriptas permitieron determinar la estrategia y planificación del abordaje quirúrgico obteniéndose una excelente correlación entre los hallazgos imagenológicos e intraoperatorios.<br />
Conclusión. En tumores cerebrales localizados en áreas elocuentes los estudios de RMI con técnicas especiales pueden contribuir sustancialmente a: 1. El diagnóstico presuntivo del tipo de tumor, grado de malignidad e infiltración del mismo. 2. La planificación de la estrategia quirúrgica para minimizar las secuelas neurológicas post operatorias.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Carlos Morales]]></dcterms:creator>
    <dcterms:creator><![CDATA[Manuel Martínez]]></dcterms:creator>
    <dcterms:creator><![CDATA[Jorge Shilton]]></dcterms:creator>
    <dcterms:creator><![CDATA[Alberto Prosen]]></dcterms:creator>
    <dcterms:creator><![CDATA[Claudio Vázquez]]></dcterms:creator>
    <dcterms:creator><![CDATA[Nilda Goldemberg]]></dcterms:creator>
    <dcterms:creator><![CDATA[Claudio Bruno]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Horacio J. Fontana]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 2007]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/331">
    <dcterms:title><![CDATA[Técnicas microquirúrgicas usadas en la reparación de los nervios periférico (Revisión de la literatura)]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Revisión de la Literatura]]></dcterms:description>
    <dcterms:abstract><![CDATA[Los autores hacen una revisión de la literatura y de las bases de datos PubMed y Lilacs acerca de las principales técnicas microquirúgicas usadas en las operaciones de los nervios periféricos. Concluyen que este tipo de intervención quirúrgica depende del uso correcto de la técnica y también de otros factores.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Flávio Freinkel Rodrigues]]></dcterms:creator>
    <dcterms:creator><![CDATA[Rafael Orta Pérez]]></dcterms:creator>
    <dcterms:creator><![CDATA[Rafael Orta Pérez]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Graciela Zúccaro]]></dcterms:publisher>
    <dcterms:date><![CDATA[Diciembre 2009]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/941">
    <dcterms:title><![CDATA[Temporal Bone &amp; Posterior Cranial Fossa.]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Novedades Editoriales]]></dcterms:description>
    <dcterms:creator><![CDATA[León Turjanski]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Julio C. Suárez]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 1996]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/638">
    <dcterms:title><![CDATA[Teratoma del Rafe Bulbar]]></dcterms:title>
    <dcterms:description><![CDATA[Trabajos Breves]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objetive. To describe the case of a medulla oblongata benign teratoma associated with endocrinological disorders.<br />
Description. A 23 years old female patient presented with headache, vomiting, dizzines, ataxia, amenorrhea and galactorrhea, progressive during the iast 5 years. MRI showed a fourth ventricle tumoral lesion that involved the medulla oblongata with hydrocephalus.<br />
Intervention. First a ventriculo-peritoneal shunting was performed. One week later through a midline approach a microsurgical complete removal was done. Postoperative outcome was good. MRI 6 months later was free of tumor. After one year the neurological examination was normal.<br />
Conclusion. Teratomas of the medulla oblongata are rare lesions. Surgery is the treatment of choice.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Myriam C. Montenegro]]></dcterms:creator>
    <dcterms:creator><![CDATA[Norberto Ruiz de Huidobro]]></dcterms:creator>
    <dcterms:creator><![CDATA[Pantaleón Saladino]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Juan José Mezzadri]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Luis Augusto Lemme-Plaghos]]></dcterms:publisher>
    <dcterms:date><![CDATA[Septiembre 2004]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[1. Greenberg, M. Handbook of Neurosurgery, 5a. edition. Thieme (New York) 2001; p. 455.<br />
<br />
2. Youmans, J. Editor. Neurological Surgery 4a. ed, WB Saunders. 1996; Vol 4, cap. 115.<br />
<br />
3. Tsuzuki N, Kato H, Ishihara S, Miyazawa T, Nawashuro H, Shima K. Malgnant teratoma of the medulla oblongata in an adult male. Acta Neurochir (Wien) 2001; 143: 1303-4]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/1107">
    <dcterms:title><![CDATA[Teratoma del Tercer Ventrículo. A propósito de un caso]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Reporte de Caso]]></dcterms:description>
    <dcterms:creator><![CDATA[M. O. Lopez Ferro]]></dcterms:creator>
    <dcterms:creator><![CDATA[J. L. Niño]]></dcterms:creator>
    <dcterms:creator><![CDATA[J. Muñiz De Igneson]]></dcterms:creator>
    <dcterms:creator><![CDATA[P. Ruiz Barnes]]></dcterms:creator>
    <dcterms:creator><![CDATA[J. R. Boixados Servat]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Hugo N. Usarralde]]></dcterms:publisher>
    <dcterms:date><![CDATA[Agosto 1985]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/204">
    <dcterms:title><![CDATA[TERCER VENTRICULOSTOMÍA ENDOSCÓPICA EN PACIENTES ADULTOS]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo Original]]></dcterms:description>
    <dcterms:abstract><![CDATA[<p><strong>RESUMEN</strong><br /> <strong>Objetivo.</strong> Describir las indicaciones, ilustrar la t&eacute;cnica quir&uacute;rgica y analizar los resultados de la tercer ventriculostom&iacute;a endosc&oacute;pica (TVE) en el manejo de la hidrocefalia de distinta etiolog&iacute;a, exclusivamente en poblaci&oacute;n adulta, en nuestra serie de 50 casos consecutivos.<br /> <strong>Material y m&eacute;todo.</strong> De un total de 79 procedimientos endosc&oacute;picos intraventriculares durante el per&iacute;odo agosto 2005 - octubre 2011 fueron realizadas 50 TVE solamente en poblaci&oacute;n adulta. Las indicaciones fueron: 23 tumores, 8 hidrocefalias normotensivas del adulto, 7 estenosis acueductales, 3 ventriculomegalias de larga data del adulto, 3 neurocisticercosis, 2 hematomas de fosa posterior, 2 infartos cerebelosos y 2 quistes aracnoideos.<br /> <strong>Resultados.</strong> La edad promedio fue de 51 a&ntilde;os (rango de 21-78 a&ntilde;os). Sexo: 28 hombres y 22 mujeres. Se realizaron 39 cirug&iacute;as en calidad programada y 11 en calidad de urgencia. El tiempo de estad&iacute;a hospitalaria promedio fue de 2.5 d&iacute;as. El tiempo de seguimiento fue de 3 a 58 meses (media de 24 meses). El &eacute;xito cl&iacute;nico de la TVE se constat&oacute; en 38 de 50 casos (76%). La mejor&iacute;a de la hidrocefalia en los controles por im&aacute;genes se confirm&oacute; en 25 casos (50%). La morbilidad operatoria estuvo presente en cuatro casos (8 %): 1 meningitis, 1 pneumoenc&eacute;falo a tensi&oacute;n, 1 paresia de nervio motor ocular externo transitoria, 1 hemorragia ventricular asociada a biopsia tumoral. Siete casos (14%) requirieron una v&aacute;lvula de derivaci&oacute;n, siendo la mayor&iacute;a por progresi&oacute;n tumoral.<br /> <strong>Conclusi&oacute;n.</strong> La TVE es una t&eacute;cnica f&aacute;cilmente reproducible que representa el gold standard actual en el manejo de una gran variedad de hidrocefalias obstructivas, aunque sus indicaciones se han extendido tambi&eacute;n a algunas hidrocefalias comunicantes. En nuestra experiencia &uacute;nicamente con pacientes adultos, la TVE result&oacute; ser el procedimiento de elecci&oacute;n para el tratamiento definitivo de la hidrocefalia en la mayor&iacute;a de los casos. Debe considerarse siempre a la evoluci&oacute;n cl&iacute;nica favorable como el elemento principal para determinar el &eacute;xito del procedimiento y secundariamente a los estudios por im&aacute;genes.<br /> <strong>Palabras clave:</strong> hidrocefalia, neuroendoscop&iacute;a, tercer ventriculostom&iacute;a endosc&oacute;pica.</p>]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Santiago González Abbatil]]></dcterms:creator>
    <dcterms:creator><![CDATA[Alejandro Szkope]]></dcterms:creator>
    <dcterms:creator><![CDATA[Paula Ypa]]></dcterms:creator>
    <dcterms:creator><![CDATA[Federico Fernández Molina]]></dcterms:creator>
    <dcterms:creator><![CDATA[Rafael Torino]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Rafael Torino]]></dcterms:publisher>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:language><![CDATA[Inglés]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[<p><strong>Bibliograf&iacute;a</strong></p>
<ol>
<li>Kadrian D, van Gelder J, Florida D, et al. Long-term reliabilityof endoscopic third ventriculostomy. Neurosurgery 2008; 62:614-21.</li>
<li>Sacko O, Boetto S, Lauwers-Cances V, Dupuy M, Roux F.E. Endoscopic third ventriculostomy: outcome analysis in 368 procedures. J Neurosurg Pediatr 2010; 5: 68-74.</li>
<li>Javadpour M, Mallucci C. The role of neuroendoscopy in the management of tectal gliomas. Childs Nerv Syst 2004; 20:852-7.</li>
<li>Jenkinson M, Hayhurst C, Al-Jumaily M, Kandasamy J, Clark S, Mallucci C, The role of endoscopic third ventriculostomy in adult patients with hydrocephalus. J Neurosurg 2009; 110:861-6.</li>
<li>Amini A, Schmidt RH. Endoscopic third ventriculostomy in aseries of 36 adult patients. Neurosurg Focus 2005; 19: E9.</li>
<li>Fritsch MJ, Doerner L, Kienke S, Mehdorn HM. Hydrocephalus in children with posterior fossa tumors: role of endoscopic third ventriculostomy. J Neurosurg 2005; 103: 40-2.</li>
<li>Fritsch M.J, Kienke S, Ankermann T, Padoin M, Mehdom H.M. Endoscopic third ventriculostomy in infants. J Neurosurg 2005; 103: 50-3.</li>
<li>Hellwig D, Grotenhuis J.A, Tirakotai W, et al. Endoscopic third ventriculostomy for obstructive hydrocephalus. Neurosurg Rey 2005; 28:1-34.</li>
<li>Li K.W, Roonprapunt C, Lawson H.C, Abbott IR, WisoffJ, Epstein F. Endoscopic third ventriculostomy for hydrocephalus associatedwith tectal gliomas. Neurosurg Focus 2005; 18: E2.</li>
<li>O'Brien D.F, Hayhurst C, Pizer B, Mallucci C.L. Outcomes in patients undergoing single-trajectory endoscopic third ventriculostomy and endoscopic biopsy for midline tumors presenting with obstructive hydrocephalus. J Neurosurg 2006; 105:219-26.</li>
<li>Siomin V, Cinalli G, Grotenhuis A, et al. Endoscopic third ventriculostomy in patients with cerebrospinal fluid infection and/ or hemorrhage. J Neurosurg 2002; 97: 519-24.</li>
<li>Yamini B, Refai D, Rubin C.M, Frim D.M. Initial endoscopic management of pineal region tumors and associated hydrocephalus: clinical series and literature review. J Neurosurg 2004;100: 437-41.</li>
<li>Fukuhara T, Luciano MG. Clinical features of late-onset idiopathic aqueductal stenosis. Surg Neurol 2001; 55: 132-37.</li>
<li>Hayhurst C, Osman-Farah J, Das K, Mallucci C.L. Initial management of hydrocephalus associated with Chiari 1-syringomyelia complex by endoscopic third ventriculostomy: an outcome analysis. J Neurosurg 2008; 108: 1211-4.</li>
<li>Hopf N.J, Grunert P, Fries G, Resch KD, Pemeczky A. Endoscopic third ventriculostomy: outcome analysis of 100 consecutive procedures. Neurosurgery 1999; 44:795-6.</li>
<li>Mohanty A, Suman R, Shankar SR, Satish S, Praharaj SS. Endoscopic third ventriculostomy in the management of Chiari I malformation and syringomyelia associated with hydrocephalus. Clin Neurol Neurosurg 2005; 108: 87-92.</li>
<li>O'Brien D.F, Javadpour M, Collins D.R, Spennato P, Mallucci C.L. Endoscopic third ventriculostomy: an outcome analysis of primary cases and procedures performed after ventriculoperitonealshunt malfunction. J Neurosurg 2005; 103: 393-400.</li>
<li>Rahme R, Hourani R, Moussa R, et al. Endoscopic third ventri culostomy: the Lebanese experience. Pediatr Neurosurg 2009; 45: 361-7.</li>
<li>Sainte-Rose C, Cinalli G, ROUX FE, Maixner R, Chumas PD, Mansour M. Management of hydrocephalus in pediatric patients with posterior fossa tumors: the role of endoscopic third ventriculostomy. J Neurosurg 2001; 95: 791-7.</li>
<li>Tisell M, Almstrom O, Stephensen H, Tullberg M, Wikkelso C. How effective is endoscopic third ventriculostomy in treating adult hydrocephalus caused by primary aqueductal stenosis? Neurosurgery 2000; 46:104-10.</li>
<li>Cage TA, Auguste KI, Wrensch M, Wu YW, Gupta N. Self-reported functional outcome after surgical intervention in patients with idiopathic normal pressure hydrocephalus. J Clin Neurosci 2011; 18:649-54.</li>
<li>Gangemi M, Maiuri F, Buonamassa S, Colella G, de Divitiis E. Endoscopic third ventriculostomy in idiopathic normal pressure hydrocephalus. Neurosurgery 2004; 55: 129-34.</li>
<li>Gangemi M, Maiuri F, Naddeo M, et al. Endoscopic third ventriculostomy in idiopathic normal pressure hydrocephalus: an Italian multicenter study. Neurosurgery 2008; 63: 62-7.</li>
<li>Di S, Shimoda M, Shibata M, Honda Y, Togo K, Shinoda M. Pathophysiology of long-standing overt ventriculomegaly in adults. J Neurosurg 2000; 92: 933-40.</li>
<li>Rekate HL. Longstanding overt ventriculomegaly in adults: pitfalls in treatment with endoscopic third ventriculostomy. Neurosurg Focus 2007; 22: E6.</li>
<li>Dirwer A, Yildiz E, Kohan S, Memet &Uacute;zek M. Analysis of endoscopic third ventriculostomy patency by MRI: value of different pulse sequences, the sequence parameters, and the imaging planes for investigation of flow void. Childs Nerv Syst 2011; 27: 127-35.</li>
<li>Stivaros SM, Sinclair D, Bromiley PA, Kim J, Thorne J, Jackson A. Endoscopic third ventriculostomy: predicting outcome with phase-contrast MR imaging. Radiology 2009; 252: 825-32.</li>
<li>Beems T, Grotenhuis J.A. Is the success rate of endoscopic third ventriculostomy age-dependent? An analysis of the results of endoscopic third ventriculostomy in young children. Childs Nerv Syst 2002; 18: 605-8.</li>
<li>Gangemi M, Donati P, Maiuri F, et al. Endoscopic third ventriculostomy for hydrocephalus. Minim Invasive Neurosurg1999; 42: 128-32.</li>
<li>Z&uacute;ccaro G, Portillo S, Fernandez Molina G, et al: Diferentes t&eacute;cnicas neuroendosc&oacute;picas para el tratamiento de la hidrocefalia pedi&aacute;trica. Estudio multic&eacute;ntrico en 360 pacientes. Rey Argent Neurocir 2003; 17:158-60.</li>
<li>Baldauf J, Oertel J, Gaab MR, Schroeder HW. Endoscopic third ventriculostomy for occlusive hydrocephalus caused by cerebellar infarction. Neurosurgery 2006; 59: 53944.</li>
<li>Roux FE, Boetto S, Tremoulet M. Third ventriculocisternostomy in cerebellar haematomas: Acta Neurochir (Wien) 2002; 144: 337-42.</li>
<li>Schroeder HW, Niendorf WR, Gaab MR. Complications of endoscopic third ventriculostomy. J Neurosurg 2002; 96: 1032-40.</li>
<li>Bouras T, Sgouros S. Complications of endoscopic third ventriculostomy. J Neurosurg Pediatr 2011; 7:643-9.</li>
</ol>]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/438">
    <dcterms:title><![CDATA[Tercer ventriculostomía endoscópica: mecanismos de fracaso y consideraciones sobre refenestración o colocación de Shunt]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Trabajos Breves]]></dcterms:description>
    <dcterms:abstract><![CDATA[Objective: To evaluate retrospectively, a group of patients of our series, that had a failed ventriculostomy to try to determine the causes and the treatement offered to them.<br />
Material and method: 140 ETVs were performed in 132 patients; 120 of them are shunt free (90.90%). Eight patients (5.71%) showed closure of the ventriculostomy: in 4, it was presumed to be related to postoperative radiotherapy; 2 patients had an insufficient opening of the Lilliequist membrane and 2 showed ostoma closure of unknown origin. In all of them a second ETV was performed, and the procedure was successful. Twelve patients (8.57%) required shunt placement; 4, with a history of septated postmeningitis hydrocephalus, now only need a single ventricular catheter. Of the remaining, 3 presented with meningeal seeding from malignant tumors; 1 with racemous neurocysticercosis; 1 with multiple malformations and history meningitis; 2 with a previously unknown aresorptive component; 1 with history of post-shunt meningitis. Most patients with ETV failure developed CSF fistula. In all these cases, patency of the ostoma was confirmed during re-exploration, and consequently, a shunt was indicated.<br />
Discussion and Conclusions: We consider ETV to be the standard treatment for obstructive hydrocephalus. With low morbidity in our series (4.68%) and no mortality. Re-exploration and eventual re-fenestration are indicated in all cases of ETV failure, given the benefits of shunt independence.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Carlos E. Gagliardi]]></dcterms:creator>
    <dcterms:creator><![CDATA[Luis M. Cuello]]></dcterms:creator>
    <dcterms:creator><![CDATA[Patricia Z. Maggiora]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Horacio J. Fontana]]></dcterms:publisher>
    <dcterms:date><![CDATA[Septiembre 2007]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/97">
    <dcterms:title><![CDATA[test]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículo de Revisión]]></dcterms:description>
    <dcterms:abstract><![CDATA[test]]></dcterms:abstract>
    <dcterms:tableOfContents><![CDATA[test]]></dcterms:tableOfContents>
    <dcterms:creator><![CDATA[test]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Marcelo Platas]]></dcterms:publisher>
    <dcterms:date><![CDATA[test]]></dcterms:date>
    <dcterms:dateAccepted><![CDATA[test]]></dcterms:dateAccepted>
    <dcterms:dateSubmitted><![CDATA[test]]></dcterms:dateSubmitted>
    <dcterms:issued><![CDATA[test]]></dcterms:issued>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:references><![CDATA[test]]></dcterms:references>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[test]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/980">
    <dcterms:title><![CDATA[The 1st Interim Meeting of the European Society for Paediatric Neurosurgery (ESPN)<br />
Eilat, Israel, Marzo 19-23, 1995.]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Reseña de Congreso]]></dcterms:description>
    <dcterms:creator><![CDATA[Edgardo Schijman]]></dcterms:creator>
    <dcterms:publisher><![CDATA[León Turjanski]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Julio C. Suárez]]></dcterms:publisher>
    <dcterms:date><![CDATA[Junio 1995]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/602">
    <dcterms:title><![CDATA[The International Subarachnoidal Aneurysm Trial (ISAT)]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Reseña de Congreso]]></dcterms:description>
    <dcterms:creator><![CDATA[James V. Byrne]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Luis Augusto Lemme-Plaghos]]></dcterms:publisher>
    <dcterms:date><![CDATA[Diciembre 2004]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Inglés]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/726">
    <dcterms:title><![CDATA[Tiempos de Cambio: La Revista en la Web]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Editorial]]></dcterms:description>
    <dcterms:creator><![CDATA[Luis Lemme-Plaghos <br />
]]></dcterms:creator>
    <dcterms:creator><![CDATA[Juan José Mezzadri ]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Luis Augusto Lemme-Plaghos]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Juan José Mezzadri]]></dcterms:publisher>
    <dcterms:date><![CDATA[Marzo 2003]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/612">
    <dcterms:title><![CDATA[Tomografía de coherencia óptica como predictor de recuperación visual en pacientes con macroadenomas hipofisarios]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Trabajos Premiados]]></dcterms:description>
    <dcterms:abstract><![CDATA[Introducción: Los macroadenomas hipofisarios tienen una elevada incidencia de pérdida visual como consecuencia de la compresión de la vía óptica. La tomografía de coherencia óptica (TCO) ha sido descripta como un factor pronóstico de recuperación. Nuestro objetivo es evaluar si la TCO puede predecir los resultados de recuperación visual.<br />
Material y Métodos: Se realizó un trabajo prospectivo de macroadenomas hipofisarios con compresión de la vía visual e indicación quirúrgica entre febrero del 2015 y noviembre del 2016. Se evaluaron los estudios de agudeza visual, perimetría computarizada y TCO. Se compararon los resultados tras 6-12 semanas y 6-9 meses de la intervención. Las variables cuantitativas continuas dependientes e independientes se relacionaron mediante una prueba T de Student, regresión lineal y correlación de Pearson considerando como estadísticamente significativo una p&lt;0,05. <br />
Resultados: La alteración en la agudeza visual y la perimetría computarizada preoperatorios, fue mayor en aquellos pacientes con TCO alterada. Tanto los pacientes con CFNR normal como adelgazada se constató una mejoría significativa del campo visual a las 6-12 semanas. Los valores de TCO se correlacionaron en forma inversa con los valores de campo visual, lo que significa que a mayor grosor menor es el déficit campimétrico. Los pacientes con TCO normal tuvieron una mejor recuperación ya que el defecto campimétrico preoperatorio fue más leve. La correlación entre los valores de TCO y la mejoría del campo visual no fue significativa, lo que sugiere independientemente del grosor del nervio los pacientes mejoran el campo visual tras la cirugía. <br />
Conclusión: El campo visual computarizado continúa siendo el estudio de elección en la evaluación de pacientes con macroadenomas. La TCO no se considera un predictor de outcome visual.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Sofía Beltrame]]></dcterms:creator>
    <dcterms:creator><![CDATA[ Jorge Rasmussen]]></dcterms:creator>
    <dcterms:creator><![CDATA[Pedro Plou]]></dcterms:creator>
    <dcterms:creator><![CDATA[Moira Altszul]]></dcterms:creator>
    <dcterms:creator><![CDATA[Claudio Yampolsky]]></dcterms:creator>
    <dcterms:creator><![CDATA[Pablo Ajler]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Mariano Socolovsky]]></dcterms:publisher>
    <dcterms:date><![CDATA[Diciembre 2017]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
    <dcterms:bibliographicCitation><![CDATA[Anderson D, Faber P, Marcovitz S, et al. Pituitary tumors and the ophthalmologist. Ophthalmology, 1983; 90, 1265–70.<br />
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Danesh-Meyer H, Papchenko T, Savino P, et al. In vivo retinal nerve fiber layer thickness measured by optical coherence tomography predicts visual recovery after surgery for parachiasmal tumors. Invest Ophthalmol Vis Sci, 2008; 49, 1879–85.<br />
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Jacob M, Raverot G, Jouanneau E, et al. Predicting visual outcome after treatment of pituitary adenomas with optical coherence tomography. Am J Ophthalmol 2009;147, 64–70.<br />
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Marcus M, Vitale S, Calvert PC, Miller NR. Visual parameters in patients with pituitary adenoma before and after transsphenoidal surgery. Aust NZJ Ophthalmol 1991; 19, 111–118.<br />
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Monteiro ML, Zambon BK, Cunha LP. Predictive factors for the development of visual loss in patients with pituitary macroadenomas and for visual recovery after optic pathway decompression. Can J Ophthalmol 2010; 45, 404–8.<br />
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Moon C, Hwang S, Ohn Y, et al. The time course of visual field recovery and changes of retinal ganglion cells after optic chiasmal decompression. Invest Ophthalmol Vis Sci, 2011; 52, 7966–73.<br />
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Punjabi O, Stamper R, Bostrom A, Han Y, Lin SC. Topographic comparison of the visual function on multifocal visual evoked potentials with optic nerve structure on Heidelberg retinal tomography. Ophthalmology, 2008: 115(3), 440–446.<br />
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Qiao N, Zhang Y, Ye Z, Shen M, Shou X, et al. Comparison of mulifocal visual evoked potencial, static automated perimetry, and optical coherence tomography findings for assesing visal pathways in patients with pituitary adenomas. Pituitary 2015; 18, 598-603.<br />
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Sullivan LJ, O’Day J, McNeill P. Visual outcomes of pituitary adenoma surgery. St. Vincent’s Hospital 1968–1987. J Clin Neuroophthalmol 1991; 11, 262–267.<br />
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Yoneoka Y, Hatase T, Watanabe N, et al. Early morphological recovery of the optic chiasm is associated with excellent visual outcome in patients with compressive chiasmal syndrome caused by pituitary tumors. Neurol Re 2015; 37, 1–8.]]></dcterms:bibliographicCitation>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/665">
    <dcterms:title><![CDATA[Toxina Botulínica en Parálisis Cerebral Infantil]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Artículos Varios]]></dcterms:description>
    <dcterms:abstract><![CDATA[La lesión de la neurona motora superior en la parálisis cerebral infantil produce una perdida de la inhibición supraespinal sobre las motoneuronas del asta anterior de la médula espinal. La manifestación clínica predominante del síndrome de la neurona motora superior es la espasticidad . Esta espasticidad conduce a una discapacidad permanente, aunque no estable, para el movimiento. La toxina botulínica inhibe la contracción muscular previniendo la liberación de las vesículas con acetilcolina a nivel de los terminales presinápticos de la placa neuromuscular. La inyección intramuscular de toxina botulínica es un método eficaz y bien tolerado para el manejo de la espasticidad en pacientes con parálisis cerebral.]]></dcterms:abstract>
    <dcterms:creator><![CDATA[Mario Lampropulos]]></dcterms:creator>
    <dcterms:creator><![CDATA[Miguel Puigdevall]]></dcterms:creator>
    <dcterms:publisher><![CDATA[Edgardo Schijman]]></dcterms:publisher>
    <dcterms:publisher><![CDATA[Hugo Pomata]]></dcterms:publisher>
    <dcterms:date><![CDATA[Mayo 2004]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/260">
    <dcterms:title><![CDATA[Trabajos de Presentación de Panel]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Resúmenes]]></dcterms:description>
    <dcterms:publisher><![CDATA[Rafael Torino]]></dcterms:publisher>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/373">
    <dcterms:title><![CDATA[Trabajos de presentación en panel]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Resúmenes]]></dcterms:description>
    <dcterms:date><![CDATA[Octubre 2008]]></dcterms:date>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/384">
    <dcterms:title><![CDATA[Trabajos de presentación en panel]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Resúmenes]]></dcterms:description>
    <dcterms:publisher><![CDATA[Horacio J. Fontana]]></dcterms:publisher>
    <dcterms:date><![CDATA[Julio 2008]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/427">
    <dcterms:title><![CDATA[Trabajos de presentación en panel]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Resúmenes]]></dcterms:description>
    <dcterms:publisher><![CDATA[Horacio J. Fontana]]></dcterms:publisher>
    <dcterms:date><![CDATA[Diciembre 2007]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/259">
    <dcterms:title><![CDATA[Trabajos de Presentación Oral]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Resúmenes]]></dcterms:description>
    <dcterms:publisher><![CDATA[Rafael Torino]]></dcterms:publisher>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/374">
    <dcterms:title><![CDATA[Trabajos de presentación oral]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Resúmenes]]></dcterms:description>
    <dcterms:publisher><![CDATA[Horacio J. Fontana]]></dcterms:publisher>
    <dcterms:date><![CDATA[Octubre 2008]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description><rdf:Description rdf:about="https://aanc.org.ar/ranc/items/show/385">
    <dcterms:title><![CDATA[Trabajos de presentación oral]]></dcterms:title>
    <dcterms:subject><![CDATA[Neurocirugía]]></dcterms:subject>
    <dcterms:description><![CDATA[Resúmenes]]></dcterms:description>
    <dcterms:publisher><![CDATA[Horacio J. Fontana]]></dcterms:publisher>
    <dcterms:date><![CDATA[Julio 2008]]></dcterms:date>
    <dcterms:rights><![CDATA[Asociación Argentina de Neurocirugía]]></dcterms:rights>
    <dcterms:language><![CDATA[Español]]></dcterms:language>
</rdf:Description></rdf:RDF>
